Molecular Epidemiology of Bacteremia: Association of Molecular Factors With the Source of Infection

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Molecular Epidemiology of Bacteremia: Association of Molecular Factors With the Source of Infection

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im Januar 2018 veröffentlichter wissenschaftlicher Artikel

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scientific article published on 25 September 2018

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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Molecular Epidemiology of Bact ...... s With the Source of Infection

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Molecular Epidemiology of Bact ...... s With the Source of Infection

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Molecular Epidemiology of Bact ...... s With the Source of Infection

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Molecular Epidemiology of Bact ...... s With the Source of Infection

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Molecular Epidemiology of Bact ...... s With the Source of Infection

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Molecular Epidemiology of Bact ...... s With the Source of Infection

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Molecular Epidemiology of Stap ...... s With the Source of Infection

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Benito Almirante

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Carmen Gómez-González

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Dafne Pérez-Montarelo

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Enrique Ruiz de Gopegui

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Esther Viedma

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Fernando Chaves

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Irene Muñoz-Gallego

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Nuria Fernández-Hidalgo

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Rafael San Juan

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P2860

Mortality after Staphylococcus aureus bacteraemia in two hospitals in Oxfordshire, 1997-2003: cohort study

Staphylococcus aureus Bloodstream Infection and Endocarditis--A Prospective Cohort Study

Antimicrobial-Resistant Pathogens Associated With Healthcare-Associated Infections: Summary of Data Reported to the National Healthcare Safety Network at the Centers for Disease Control and Prevention, 2011-2014.

Molecular epidemiology of Staphylococcus aureus bacteremia in a single large Minnesota medical center in 2015 as assessed using MLST, core genome MLST and spa typing

Population studies of methicillin-resistant and -sensitive Staphylococcus aureus strains reveal a lack of variability in the agrD gene, encoding a staphylococcal autoinducer peptide.

Relationships between Staphylococcus aureus genetic background, virulence factors, agr groups (alleles), and human disease

Association between Staphylococcus aureus strains carrying gene for Panton-Valentine leukocidin and highly lethal necrotising pneumonia in young immunocompetent patients.

The innate immune modulators staphylococcal complement inhibitor and chemotaxis inhibitory protein of Staphylococcus aureus are located on beta-hemolysin-converting bacteriophages.

Staphylococcus aureus β-toxin production is common in strains with the β-toxin gene inactivated by bacteriophage.

Characterization of alpha-toxin hla gene variants, alpha-toxin expression levels, and levels of antibody to alpha-toxin in hemodialysis and postsurgical patients with Staphylococcus aureus bacteremia.

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2210

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scholarly article

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10.3389/FMICB.2018.02210

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English

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María Nieves Larrosa

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2018-09-25T00:00:00Z

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30319561

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6167439

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