TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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P1476

TNIP1 Regulates -Induced Innate Immune Functions in Epidermal Keratinocytes

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Beáta Szilvia Bolla

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Gábor Tax

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Kornélia Szabó

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Lajos Kemény

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Lilla Erdei

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Renáta Bozó

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P2860

ABIN-1 binds to NEMO/IKKgamma and co-operates with A20 in inhibiting NF-kappaB

The use of isotretinoin in acne

The skin microbiome

ABIN-1 is a ubiquitin sensor that restricts cell death and sustains embryonic development

The zinc finger protein A20 inhibits TNF-induced NF-kappaB-dependent gene expression by interfering with an RIP- or TRAF2-mediated transactivation signal and directly binds to a novel NF-kappaB-inhibiting protein ABIN

Innate immune signaling in defense against intestinal microbes

A20-Binding Inhibitor of NF-κB Activation 1 is a Physiologic Inhibitor of NF-κB: A Molecular Switch for Inflammation and Autoimmunity

Propionibacterium acnes strain populations in the human skin microbiome associated with acne

A20-binding inhibitor of NF-κB (ABIN1) controls Toll-like receptor-mediated CCAAT/enhancer-binding protein β activation and protects from inflammatory disease

Negative regulation of toll-like receptor-mediated immune responses

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2155

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scholarly article

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10.3389/FIMMU.2018.02155

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English

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Edit Urbán

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2018-01-01T00:00:00Z

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30319618

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6165910

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