about
Secondary replicative function of CD8+ T cells that had developed an effector phenotypeTumoral immune suppression by macrophages expressing fibroblast activation protein-α and heme oxygenase-1.Depletion of stromal cells expressing fibroblast activation protein-α from skeletal muscle and bone marrow results in cachexia and anemia.Targeting CXCL12 from FAP-expressing carcinoma-associated fibroblasts synergizes with anti-PD-L1 immunotherapy in pancreatic cancer.Notch2 is required for the proliferation of cardiac neural crest-derived smooth muscle cells.CD27 mediates interleukin-2-independent clonal expansion of the CD8+ T cell without effector differentiation.Pathways of memory CD8+ T-cell development.Suppression of antitumor immunity by stromal cells expressing fibroblast activation protein-alpha.BCL-6 is expressed in breast cancer and prevents mammary epithelial differentiation.Cutting edge: Virus-specific CD8+ T cell clones and the maintenance of replicative function during a persistent viral infection.Generation of mice that conditionally express the activation domain of Notch2.A common region of 10p deleted in DiGeorge and velocardiofacial syndromesFunctional conservation of Notch1 and Notch2 intracellular domainsThe protein phosphatase 2A B56γ regulatory subunit is required for heart development
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-5040-0067
@en
name
Matthew Kraman
@ast
Matthew Kraman
@en
Matthew Kraman
@es
Matthew Kraman
@nl
type
label
Matthew Kraman
@ast
Matthew Kraman
@en
Matthew Kraman
@es
Matthew Kraman
@nl
prefLabel
Matthew Kraman
@ast
Matthew Kraman
@en
Matthew Kraman
@es
Matthew Kraman
@nl
P106
P31
P496
0000-0002-5040-0067