COX-2 is not required for the development of murine chronic pancreatitis
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The effect of sulindac, a non-steroidal anti-inflammatory drug, attenuates inflammation and fibrosis in a mouse model of chronic pancreatitisCannabinoid receptor 2 agonist attenuates pain related behavior in rats with chronic alcohol/high fat diet induced pancreatitisQuantitative Evaluation and Selection of Reference Genes for Quantitative RT-PCR in Mouse Acute Pancreatitis.Animal models of gastrointestinal and liver diseases. Animal models of visceral pain: pathophysiology, translational relevance, and challenges.Ibuprofen and diclofenac treatments reduce proliferation of pancreatic acinar cells upon inflammatory injury and mitogenic stimulation.Class I histone deacetylase inhibition improves pancreatitis outcome by limiting leukocyte recruitment and acinar-to-ductal metaplasia.p21(WAF1) (/Cip1) limits senescence and acinar-to-ductal metaplasia formation during pancreatitis.Enhanced proliferation of pancreatic acinar cells in MRL/MpJ mice is driven by severe acinar injury but independent of inflammation.
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COX-2 is not required for the development of murine chronic pancreatitis
description
article
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в червні 2011
@uk
name
COX-2 is not required for the development of murine chronic pancreatitis
@en
COX-2 is not required for the development of murine chronic pancreatitis
@nl
type
label
COX-2 is not required for the development of murine chronic pancreatitis
@en
COX-2 is not required for the development of murine chronic pancreatitis
@nl
prefLabel
COX-2 is not required for the development of murine chronic pancreatitis
@en
COX-2 is not required for the development of murine chronic pancreatitis
@nl
P2093
P2860
P356
P1476
COX-2 is not required for the development of murine chronic pancreatitis
@en
P2093
Achim Weber
Alberto Silva
Martha Bain
Mathias Heikenwalder
Sabrina Sonda
Theresia Reding
P2860
P304
P356
10.1152/AJPGI.00497.2010
P50
P577
2011-06-01T00:00:00Z