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Sildenafil ameliorates left ventricular T-tubule remodeling in a pressure overload-induced murine heart failure modelAutoTT: automated detection and analysis of T-tubule architecture in cardiomyocytesOverexpression of junctophilin-2 does not enhance baseline function but attenuates heart failure development after cardiac stress.Ablation of the GNB3 gene in mice does not affect body weight, metabolism or blood pressure, but causes bradycardia.β-Adrenergic receptor antagonists ameliorate myocyte T-tubule remodeling following myocardial infarction.The ryanodine receptor store-sensing gate controls Ca2+ waves and Ca2+-triggered arrhythmiasThe cardiac ryanodine receptor luminal Ca2+ sensor governs Ca2+ waves, ventricular tachyarrhythmias and cardiac hypertrophy in calsequestrin-null mice.In situ single photon confocal imaging of cardiomyocyte T-tubule system from Langendorff-perfused heartsSildenafil prevents and reverses transverse-tubule remodeling and Ca(2+) handling dysfunction in right ventricle failure induced by pulmonary artery hypertension.Carvedilol and its new analogs suppress arrhythmogenic store overload-induced Ca2+ releaseMolecular Determinants of Calpain-dependent Cleavage of Junctophilin-2 Protein in Cardiomyocytes.Novel insights on the relationship between T-tubular defects and contractile dysfunction in a mouse model of hypertrophic cardiomyopathy.Phospholamban knockout breaks arrhythmogenic Ca²⁺ waves and suppresses catecholaminergic polymorphic ventricular tachycardia in mice.Microtubule-mediated defects in junctophilin-2 trafficking contribute to myocyte transverse-tubule remodeling and Ca2+ handling dysfunction in heart failure.MG53 is dispensable for T-tubule maturation but critical for maintaining T-tubule integrity following cardiac stress.Regional distribution of T-tubule density in left and right atria in dogs.Cholesterol is required for maintaining T-tubule integrity and intercellular connections at intercalated discs in cardiomyocytesCa2+ removal mechanisms in mouse embryonic stem cell-derived cardiomyocytes.MicroRNA: a toolkit fine-tuning the dyadic "fuzzy space"?T-tubule remodeling during transition from hypertrophy to heart failure.Transient activation of PKC results in long-lasting detrimental effects on systolic [Ca2+]i in cardiomyocytes by altering actin cytoskeletal dynamics and T-tubule integrity.E-C coupling structural protein junctophilin-2 encodes a stress-adaptive transcription regulatorPreservation of the pHi during ischemia via PKC by intermittent hypoxia
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description
onderzoeker
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researcher ORCID ID = 0000-0002-5551-2279
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36514629000
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0000-0002-5551-2279