Correlation Between Caspase-3 Activation and Three Different Markers of DNA Damage in Neonatal Cerebral Hypoxia-Ischemia
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Diet-induced effects on neuronal and glial elements in the middle-aged rat hippocampusMorphological characteristics of eosinophilic neuronal death after transient unilateral forebrain ischemia in Mongolian gerbils.Hypothermic neuroprotection.Apoptotic mechanisms in the immature brain: involvement of mitochondria.Irradiation to the young mouse brain impaired white matter growth more in females than in malesGlobal gene expression in the developing rat brain after hypoxic preconditioning: involvement of apoptotic mechanisms?Mitochondria, Bioenergetics and Excitotoxicity: New Therapeutic Targets in Perinatal Brain InjuryCell Death in the Developing Brain after Hypoxia-IschemiaAge-dependent acute interference with stem and progenitor cell proliferation in the hippocampus after exposure to 1800 MHz electromagnetic radiation.Synergistic activation of caspase-3 by m-calpain after neonatal hypoxia-ischemia: a mechanism of "pathological apoptosis"?Role of caspase-3 activation in cerebral ischemia-induced neurodegeneration in adult and neonatal brain.Caspase inhibition after neonatal ischemia in the rat brain.Neural stem cells in the subventricular zone are resilient to hypoxia/ischemia whereas progenitors are vulnerable.Determinants of central nervous system adult neurogenesis are sex, hormones, mouse strain, age, and brain region.Mice overexpressing human caspase 3 appear phenotypically normal but exhibit increased apoptosis and larger lesion volumes in response to transient focal cerebral ischaemia.Expression of signaling molecules associated with apoptosis in human ischemic stroke tissue.Acupuncture decreases ischemia-induced apoptosis and cell proliferation in dentate gyrus of gerbils.Apoptosis-inducing factor is a major contributor to neuronal loss induced by neonatal cerebral hypoxia-ischemia.Effect of long-term, postasphyxial administration of magnesium sulfate on immunostaining of microtubule-associated protein-2 and activated caspase-3 in 7-day-old rat brain.X-linked inhibitor of apoptosis protein expression after ischemic injury in the human and rat developing brain.Corticosteroids effect on caspase 3 expression in an in-vitro model of hypoxic brain cells.Vitamin C protects against ethanol and PTZ-induced apoptotic neurodegeneration in prenatal rat hippocampal neurons.Lycopene ameliorates atrazine-induced oxidative damage in adrenal cortex of male rats by activation of the Nrf2/HO-1 pathway.Death effector activation in the subventricular zone subsequent to perinatal hypoxia/ischemia
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P2860
Correlation Between Caspase-3 Activation and Three Different Markers of DNA Damage in Neonatal Cerebral Hypoxia-Ischemia
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年學術文章
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name
Correlation Between Caspase-3 ...... atal Cerebral Hypoxia-Ischemia
@en
Correlation Between Caspase-3 ...... atal Cerebral Hypoxia-Ischemia
@nl
type
label
Correlation Between Caspase-3 ...... atal Cerebral Hypoxia-Ischemia
@en
Correlation Between Caspase-3 ...... atal Cerebral Hypoxia-Ischemia
@nl
prefLabel
Correlation Between Caspase-3 ...... atal Cerebral Hypoxia-Ischemia
@en
Correlation Between Caspase-3 ...... atal Cerebral Hypoxia-Ischemia
@nl
P2860
P1476
Correlation Between Caspase-3 ...... atal Cerebral Hypoxia-Ischemia
@en
P2093
Changlian Zhu
Klas Blomgren
P2860
P304
P356
10.1046/J.1471-4159.2000.0750819.X
P407
P577
2002-01-04T00:00:00Z