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CRISPR/Cas9 mediated genome editing in ES cells and its application for chimeric analysis in miceSperm equatorial segment protein 1, SPESP1, is required for fully fertile sperm in mouseGPI-anchored protein complex, LY6K/TEX101, is required for sperm migration into the oviduct and male fertility in miceExpression of TEX101, regulated by ACE, is essential for the production of fertile mouse spermatozoaJunB is essential for IL-23-dependent pathogenicity of Th17 cells.Identification of an imprinted gene cluster in the X-inactivation center.Double strand break repair by capture of retrotransposon sequences and reverse-transcribed spliced mRNA sequences in mouse zygotesCalreticulin is required for development of the cumulus oocyte complex and female fertilityGPI-AP release in cellular, developmental, and reproductive biology.Structural and functional insights into IZUMO1 recognition by JUNO in mammalian fertilization.Genome engineering uncovers 54 evolutionarily conserved and testis-enriched genes that are not required for male fertility in mice.Generation of mutant mice by pronuclear injection of circular plasmid expressing Cas9 and single guided RNA.A GPI processing phospholipase A2, PGAP6, modulates Nodal signaling in embryos by shedding CRIPTO.A chromosome 16p13.11 microduplication causes hyperactivity through dysregulation of miR-484/protocadherin-19 signaling.Ftx is dispensable for imprinted X-chromosome inactivation in preimplantation mouse embryos.Lentiviral Vector-Mediated Complementation Restored Fetal Viability but Not Placental Hyperplasia in Plac1-Deficient Mice.Production of mouse pups from germline transmission-failed knockout chimeras.Generation of precise point mutation mice by footprintless genome modification.Human Globozoospermia-Related Gene Spata16 Is Required for Sperm Formation Revealed by CRISPR/Cas9-Mediated Mouse Models.Feasibility for a large scale mouse mutagenesis by injecting CRISPR/Cas plasmid into zygotes.Factors controlling sperm migration through the oviduct revealed by gene-modified mouse models.Live imaging of X chromosome reactivation dynamics in early mouse development can discriminate naïve from primed pluripotent stem cells.CRISPR/Cas9-based genome editing in mice by single plasmid injection.Regulation of inflammatory responses by dynamic subcellular localization of RNA-binding protein Arid5a.Induction of DNA methylation by artificial piRNA production in male germ cells.Comparison of gene expression in male and female mouse blastocysts revealed imprinting of the X-linked gene, Rhox5/Pem, at preimplantation stages.Activation of Myofibroblast TRPA1 by Steroids and Pirfenidone Ameliorates Fibrosis in Experimental Crohn's Disease.The Mg2+ transporter CNNM4 regulates sperm Ca2+ homeostasis and is essential for reproduction.Sperm calcineurin inhibition prevents mouse fertility with implications for male contraceptive.MARCKSL1 Regulates Spine Formation in the Amygdala and Controls the Hypothalamic-Pituitary-Adrenal Axis and Anxiety-Like Behaviors.Co-expression of sperm membrane proteins CMTM2A and CMTM2B is essential for ADAM3 localization and male fertility in micemDia1/3 generate cortical F-actin meshwork in Sertoli cells that is continuous with contractile F-actin bundles and indispensable for spermatogenesis and male fertilityDGKγ Knock-Out Mice Show Impairments in Cerebellar Motor Coordination, LTD, and the Dendritic Development of Purkinje Cells through the Activation of PKCγCRISPR/Cas9-Mediated Genome Editing Reveals Oosp Family Genes are Dispensable for Female Fertility in MiceSpermatozoa lacking Fertilization Influencing Membrane Protein (FIMP) fail to fuse with oocytes in miceCharacterization of a BAC transgenic mouse expressing Krt19-driven iCre recombinase in its digestive organsAn App knock-in mouse inducing the formation of a toxic conformer of Aβ as a model for evaluating only oligomer-induced cognitive decline in Alzheimer's diseaseCRISPR/Cas9-mediated genome editing reveals 30 testis-enriched genes dispensable for male fertility in mice†Identification of multiple male reproductive tract-specific proteins that regulate sperm migration through the oviduct in miceSeminal vesicle secretory protein 7, PATE4, is not required for sperm function but for copulatory plug formation to ensure fecundity†
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0001-8332-3507
@en
name
Yoshitaka Fujihara
@ast
Yoshitaka Fujihara
@en
Yoshitaka Fujihara
@es
Yoshitaka Fujihara
@nl
type
label
Yoshitaka Fujihara
@ast
Yoshitaka Fujihara
@en
Yoshitaka Fujihara
@es
Yoshitaka Fujihara
@nl
prefLabel
Yoshitaka Fujihara
@ast
Yoshitaka Fujihara
@en
Yoshitaka Fujihara
@es
Yoshitaka Fujihara
@nl
P106
P1153
11641452500
P31
P496
0000-0001-8332-3507