about
Inhibition of acute graft-versus-host disease with retention of graft-versus-tumor effects by the proteasome inhibitor bortezomibThe NF-κB RelB protein is an oncogenic driver of mesenchymal gliomaTopographic organization of sensory projections to the olfactory bulb.Ex vivo inhibition of NF-kappaB signaling in alloreactive T-cells prevents graft-versus-host disease.Nuclear factor-kappaB is an important modulator of the altered gene expression profile and malignant phenotype in squamous cell carcinoma.Tumor necrosis factor-like weak inducer of apoptosis (TWEAK) promotes glioma cell invasion through induction of NF-κB-inducing kinase (NIK) and noncanonical NF-κB signalingNIK regulates MT1-MMP activity and promotes glioma cell invasion independently of the canonical NF-κB pathwayNF-kappaB mediates inhibition of mesenchymal cell differentiation through a posttranscriptional gene silencing mechanism.Positive and negative regulation of EAAT2 by NF-kappaB: a role for N-myc in TNFalpha-controlled repression.IKK-dependent, NF-κB-independent control of autophagic gene expression.AI-06NON-CANONICAL NF-kB SIGNALING DRIVES THE AGGRESSIVE INVASIVENESS OF GLIOBLASTOMA.NIK/MAP3K14 Regulates Mitochondrial Dynamics and Trafficking to Promote Cell Invasion.Conjugation of Dasatinib with MHI-148 Has a Significant Advantageous Effect in Viability Assays for Glioblastoma Cells
P50
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P50
description
researcher ORCID ID = 0000-0001-8916-9707
@en
wetenschapper
@nl
name
R Sitcheran
@ast
R Sitcheran
@en
R Sitcheran
@nl
type
label
R Sitcheran
@ast
R Sitcheran
@en
R Sitcheran
@nl
prefLabel
R Sitcheran
@ast
R Sitcheran
@en
R Sitcheran
@nl
P106
P31
P496
0000-0001-8916-9707