Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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wetenschappelijk artikel

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наукова стаття, опублікована у вересні 2018

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Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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P1476

Myocardial reperfusion injury and oxidative stress: Therapeutic opportunities

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P2093

Abraham Ij Gajardo

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Jaime González-Montero

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Ramón Rodrigo

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Roberto Brito

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P2860

The no-reflow phenomenon: State of the art

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Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension

All-Trans Retinoic Acid Ameliorates Myocardial Ischemia/Reperfusion Injury by Reducing Cardiomyocyte Apoptosis.

Effects of a novel ascorbate-based protocol on infarct size and ventricle function in acute myocardial infarction patients undergoing percutaneous coronary angioplasty.

Oxidative stress and nuclear factor-kappaB activation: a reassessment of the evidence in the light of recent discoveries.

The oxygen trail: tissue oxygenation.

Endothelial dihydrofolate reductase: critical for nitric oxide bioavailability and role in angiotensin II uncoupling of endothelial nitric oxide synthase

Triggering and modulation of apoptosis by oxidative stress.

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74-86

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scholarly article

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10.4330/WJC.V10.I9.74

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English

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2018-09-26T00:00:00Z

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30344955

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reperfusion injury

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6189069

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