Elevation of Expression of Smads 2, 3, and 4, Decorin and TGF-βin the Chronic Phase of Myocardial Infarct Scar Healing
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Good and bad sides of TGFβ-signaling in myocardial infarctionMicroRNA-34a regulates cardiac fibrosis after myocardial infarction by targeting Smad4Activation of TGF-beta1-TAK1-p38 MAPK pathway in spared cardiomyocytes is involved in left ventricular remodeling after myocardial infarction in ratsDifferential and combined effects of cardiotrophin-1 and TGF-beta1 on cardiac myofibroblast proliferation and contractionTGFβ receptor activation enhances cardiac apoptosis via SMAD activation and concomitant NO releaseDecreased Smad 7 expression contributes to cardiac fibrosis in the infarcted rat heartA thrombospondin-1 antagonist of transforming growth factor-beta activation blocks cardiomyopathy in rats with diabetes and elevated angiotensin IIFibulin-2 deficiency attenuates angiotensin II-induced cardiac hypertrophy by reducing transforming growth factor-β signallingCell origin of human mesenchymal stem cells determines a different healing performance in cardiac regenerationMyocardin-related transcription factor-a controls myofibroblast activation and fibrosis in response to myocardial infarction.Preservation of myocardial structure is enhanced by pim-1 engineering of bone marrow cellsAdhesion proteins, stem cells, and arrhythmogenesis.Scleroderma and Smads: dysfunctional Smad family dynamics culminating in fibrosis.Transforming growth factor-beta1 (TGF-beta1): a potential recovery signal in the post-ischemic kidney.The role of TGFβ1 and LRG1 in cardiac remodelling and heart failure.Hydrogen sulfide alleviates myocardial collagen remodeling in association with inhibition of TGF-β/Smad signaling pathway in spontaneously hypertensive ratsAutophagy is a regulator of TGF-β1-induced fibrogenesis in primary human atrial myofibroblasts.Contribution of microRNA to pathological fibrosis in cardio-renal syndrome: impact of uremic toxinsRe-employment of developmental transcription factors in adult heart diseaseSmad3 as a mediator of the fibrotic response.Extensive induction of important mediators of fibrosis and dystrophic calcification in desmin-deficient cardiomyopathy.Beneficial effects of long-term administration of an oral formulation of Angiotensin-(1-7) in infarcted rats.The Treatment of Fibrosis of Joint Synovium and Frozen Shoulder by Smad4 Gene Silencing in RatsFibrosis in heart disease: understanding the role of transforming growth factor-beta in cardiomyopathy, valvular disease and arrhythmia.Role of Transforming Growth Factor-β1 and Smads Signaling Pathway in Intrauterine Adhesion.The role of TGF-beta signaling in myocardial infarction and cardiac remodelingNecroX-5 exerts anti-inflammatory and anti-fibrotic effects via modulation of the TNFα/Dcn/TGFβ1/Smad2 pathway in hypoxia/reoxygenation-treated rat heartsContribution of the anaphylatoxin receptors, C3aR and C5aR, to the pathogenesis of pulmonary fibrosis.Systematic characterization of myocardial inflammation, repair, and remodeling in a mouse model of reperfused myocardial infarction.Cathepsin S-mediated fibroblast trans-differentiation contributes to left ventricular remodelling after myocardial infarction.The role of SLRP-proteoglycans in osteosarcoma pathogenesis.Fate of undifferentiated mouse embryonic stem cells within the rat heart: role of myocardial infarction and immune suppression.Myocardial interstitial fluid inhibits proliferation and cardiomyocyte differentiation in pluripotent embryonic stem cells.Association of TGFBR2 polymorphism with risk of sudden cardiac arrest in patients with coronary artery disease.Thrombospondins in the heart: potential functions in cardiac remodeling.c-Ski, Smurf2, and Arkadia as regulators of TGF-beta signaling: new targets for managing myofibroblast function and cardiac fibrosis.The inflammatory response and cardiac repair after myocardial infarction.The actin-MRTF-SRF gene regulatory axis and myofibroblast differentiation.Hypoxia-sensitive pathways in inflammation-driven fibrosis.Advances in Toll-like receptor biology: Modes of activation by diverse stimuli.
P2860
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P2860
Elevation of Expression of Smads 2, 3, and 4, Decorin and TGF-βin the Chronic Phase of Myocardial Infarct Scar Healing
description
im März 1999 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 01 March 1999
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в березні 1999
@uk
name
Elevation of Expression of Sma ...... yocardial Infarct Scar Healing
@en
Elevation of Expression of Sma ...... yocardial Infarct Scar Healing
@nl
type
label
Elevation of Expression of Sma ...... yocardial Infarct Scar Healing
@en
Elevation of Expression of Sma ...... yocardial Infarct Scar Healing
@nl
prefLabel
Elevation of Expression of Sma ...... yocardial Infarct Scar Healing
@en
Elevation of Expression of Sma ...... yocardial Infarct Scar Healing
@nl
P2093
P356
P1476
Elevation of expression of Sma ...... yocardial infarct scar healing
@en
P2093
P304
P356
10.1006/JMCC.1998.0902
P50
P577
1999-03-01T00:00:00Z