Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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wetenschappelijk artikel

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наукова стаття, опублікована в жовтні 2018

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Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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P1476

Chronological Switch from Translesion Synthesis to Homology-Dependent Gap Repair

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P2093

Asako Isogawa

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Robert P Fuchs

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Shingo Fujii

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P2860

hRAD30 mutations in the variant form of xeroderma pigmentosum

All three SOS-inducible DNA polymerases (Pol II, Pol IV and Pol V) are involved in induced mutagenesis

DNA damage tolerance by recombination: Molecular pathways and DNA structures

The XPV (xeroderma pigmentosum variant) gene encodes human DNA polymerase eta

Gaps and forks in DNA replication: Rediscovering old models

Separation of recombination and SOS response in Escherichia coli RecA suggests LexA interaction sites

Error-prone repair DNA polymerases in prokaryotes and eukaryotes.

Uncoupling of leading- and lagging-strand DNA replication during lesion bypass in vivo.

Translesion DNA synthesis and mutagenesis in prokaryotes.

Nascent DNA processing by RecJ favors lesion repair over translesion synthesis at arrested replication forks in Escherichia coli.

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297-302

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scholarly article

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10.5487/TR.2018.34.4.297

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English

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P478

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P577

2018-10-01T00:00:00Z

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30370004

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toxicology

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6195876

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