about
Reprogramming in vivo produces teratomas and iPS cells with totipotency features.CtIP-Specific Roles during Cell Reprogramming Have Long-Term Consequences in the Survival and Fitness of Induced Pluripotent Stem CellsING proteins in cellular senescence.Common Telomere Changes during In Vivo Reprogramming and Early Stages of Tumorigenesis.Notch Inhibition Enhances Cardiac Reprogramming by Increasing MEF2C Transcriptional Activity.Reprogramming activity of NANOGP8, a NANOG family member widely expressed in cancer.The homeoprotein SIX1 controls cellular senescence through the regulation of p16INK4A and differentiation-related genes.Growth inhibition by the tumor suppressor p33ING1 in immortalized and primary cells: involvement of two silencing domains and effect of Ras.Tissue damage and senescence provide critical signals for cellular reprogramming in vivo.AAVvector-mediated in vivo reprogramming into pluripotencyThe tumor suppressor ING1 contributes to epigenetic control of cellular senescenceA spotlight on cancer researchers in Spain: new paradigms and disruptive ideas
P50
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P50
description
onderzoeker
@nl
researcher, ORCID id # 0000-0002-1462-2498
@en
name
María Abad
@ast
María Abad
@en
María Abad
@es
María Abad
@nl
type
label
María Abad
@ast
María Abad
@en
María Abad
@es
María Abad
@nl
prefLabel
María Abad
@ast
María Abad
@en
María Abad
@es
María Abad
@nl
P106
P1153
36960130200
P31
P496
0000-0002-1462-2498