about
Central orchestration of peripheral nutrient partitioning and substrate utilization: implications for the metabolic syndromeSaturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesityAdipose tissue NAPE-PLD controls fat mass development by altering the browning process and gut microbiota.Food restriction selectively increases hypothalamic orexin-B levels in lactating rats.Intestinal deletion of leptin signaling alters activity of nutrient transporters and delayed the onset of obesity in mice.ChemR23 knockout mice display mild obesity but no deficit in adipocyte differentiation.Intestinal epithelial MyD88 is a sensor switching host metabolism towards obesity according to nutritional status.Dietary triglycerides act on mesolimbic structures to regulate the rewarding and motivational aspects of feeding.Leptin and the adaptations of lactation in rodents and ruminants.Hypothalamic AgRP-neurons control peripheral substrate utilization and nutrient partitioningArcuate AgRP neurons and the regulation of energy balanceHippocampal lipoprotein lipase regulates energy balance in rodents.The hypothalamic arcuate nucleus and the control of peripheral substrates.Lactation modulates diurnal expression profiles of specific leptin receptor isoforms in the rat hypothalamus.Laforin, a dual specificity phosphatase involved in Lafora disease, regulates insulin response and whole-body energy balance in mice.Lipoprotein lipase in hypothalamus is a key regulator of body weight gain and glucose homeostasis in mice.Central CCL2 signaling onto MCH neurons mediates metabolic and behavioral adaptation to inflammation.Deletion of tumor necrosis factor-α receptor 1 (TNFR1) protects against diet-induced obesity by means of increased thermogenesis.NOV/CCN3: A New Adipocytokine Involved in Obesity-Associated Insulin Resistance.Additive effects of olanzapine and melanin-concentrating hormone agonism on energy balance.Regulation of serum leptin and its role in the hyperphagia of lactation in the rat.Peroxisome proliferator-activated receptor-gamma-mediated positive energy balance in the rat is associated with reduced sympathetic drive to adipose tissues and thyroid status.The LXCXE Retinoblastoma Protein-Binding Motif of FOG-2 Regulates Adipogenesis.Muscle expression of a malonyl-CoA-insensitive carnitine palmitoyltransferase-1 protects mice against high-fat/high-sucrose diet-induced insulin resistance.TNF-α transiently induces endoplasmic reticulum stress and an incomplete unfolded protein response in the hypothalamus.Irf5 deficiency in macrophages promotes beneficial adipose tissue expansion and insulin sensitivity during obesity.Increased levels of inflammatory plasma markers and obesity risk in a mouse model of Down syndrome.AgRP Neurons Require Carnitine Acetyltransferase to Regulate Metabolic Flexibility and Peripheral Nutrient Partitioning.Beige differentiation of adipose depots in mice lacking prolactin receptor protects against high-fat-diet-induced obesity.Myostatin is a key mediator between energy metabolism and endurance capacity of skeletal muscle.Corrigendum to "Increased levels of inflammatory plasma markers and obesity risk in a mouse model of Down syndrome" [Free Radic. Biol. Med. 114 (2018) 122-130].Lipoprotein Lipase Expression in Hypothalamus Is Involved in the Central Regulation of Thermogenesis and the Response to Cold Exposure.Prebiotics Supplementation Impact on the Reinforcing and Motivational Aspect of Feeding.Endocannabinoid and nitric oxide systems of the hypothalamic paraventricular nucleus mediate effects of NPY on energy expenditureGenetic deficiency of indoleamine 2,3-dioxygenase promotes gut microbiota-mediated metabolic healthPalatability Can Drive Feeding Independent of AgRP NeuronsAcyltransferase activities in the yolk sac membrane of the chick embryoDiurnal changes in hypothalamic neuropeptide and SOCS-3 expression: effects of lactation and relationship with serum leptin and food intakeShort-Term Consumption of Sucralose with, but Not without, Carbohydrate Impairs Neural and Metabolic Sensitivity to Sugar in HumansPostprandial Hyperglycemia Stimulates Neuroglial Plasticity in Hypothalamic POMC Neurons after a Balanced Meal
P50
Q26825538-FC24F302-B8F5-46B1-853C-1218D8E10F70Q28570529-FB0F1D81-BF53-49EA-8F2C-0174B6CD46F0Q30372594-8FF6FAD9-3DD3-4A41-B85E-48A2E38A461BQ31874744-55A50789-1B2E-42A0-A3D2-6196A0452FCAQ34072619-6BF24C0B-268E-4350-AD04-AFDC71AA38D5Q34374687-E6BA810D-F947-462C-ACF5-FA05853B7EB7Q34720051-33334CCD-CC11-446F-83ED-BEC09F5C6084Q35001300-A54DEE73-F067-4391-ACC7-52A106841E44Q35093285-2FB1A3E8-8658-4F90-97E9-01ED02324EA3Q36408527-A6381C24-D21F-494D-9E8F-D663BF50F89AQ36490506-22785D02-0A97-41E1-A4EB-964E00357B8DQ37636443-F89BD65F-D499-4762-96A8-58EC28225E83Q38254398-14CAA7B9-E12F-4C57-A7A8-6E68BC614D8AQ38351723-8335E898-C06C-4925-8022-448671CF302FQ39558633-F242EE70-C43E-4120-98B0-8C9768B202B3Q40221566-8BC1CE57-2A7D-4505-BCE8-86F35C56BB01Q40509061-82AB2363-E206-40F5-A08D-564523A586DDQ40562727-9CA471E9-EC12-40CA-9137-6E567B07DB34Q42809862-21627C8B-3803-4ECC-91A9-A4580795B33BQ43269622-302A5E2B-D239-4497-8042-81305475F233Q44294090-73ED0D84-F8D1-4ADC-94CD-8CFB2006FD56Q46792526-8390A013-EE2F-42C4-8A69-9FD6FFE80A8BQ47270771-C985C8FD-6E69-421D-8F51-E2E566BA03F2Q48055039-C055729B-5C2E-4A4A-8C4C-F53BD38F96B1Q48089945-7D7CD872-2633-4E15-A0D1-98D4193E1BC4Q48192209-AC092B03-5992-416F-B2EA-5B62A008D13AQ49665881-C1835018-66A2-437F-BA64-C120D78928E0Q49997199-A7272183-189D-43A3-96DA-B46EB1FEDE99Q50577860-C6C8324D-CF48-40AA-A1D2-F1D2953DD6DBQ51714503-9C960575-7CA0-4F3B-B0FE-A98D75DD017FQ53826481-695647F1-9F4F-4823-8003-A210AA582729Q54246492-4686C346-2F70-4012-9B4C-F9F72E01F7D8Q55072446-6B42E852-910C-4F60-86A7-AD0084A8F3A0Q57073954-D25AF607-4BA4-422F-B198-7D32CA67E405Q57295258-64461C45-0572-4416-9F6A-8742CA5B151AQ57600707-CA95A3D1-AB48-44D5-9BF4-30EE282E2A35Q73211499-84C63035-DEF8-4D82-BDD0-A2A51157372EQ80961271-FCD7926E-E45A-4679-9B89-3919890E5040Q90044467-DC4CE0CF-BDC5-4869-A344-7AF7F8420993Q90044665-AE03FCAE-FF4E-4202-AB71-5F1256C6501B
P50
description
onderzoeker
@nl
researcher, ORCID id # 0000-0002-7677-7460
@en
name
Raphaël G Denis
@ast
Raphaël G Denis
@en
Raphaël G Denis
@es
Raphaël G Denis
@nl
type
label
Raphaël G Denis
@ast
Raphaël G Denis
@en
Raphaël G Denis
@es
Raphaël G Denis
@nl
prefLabel
Raphaël G Denis
@ast
Raphaël G Denis
@en
Raphaël G Denis
@es
Raphaël G Denis
@nl
P106
P1153
7006222866
P31
P496
0000-0002-7677-7460