about
Genetic variation in KCNA5: impact on the atrial-specific potassium current IKur in patients with lone atrial fibrillationKv7.1 surface expression is regulated by epithelial cell polarizationAMP-activated protein kinase downregulates Kv7.1 cell surface expressionIKs Gain- and Loss-of-Function in Early-Onset Lone Atrial FibrillationPKC and AMPK regulation of Kv1.5 potassium channelsProtein kinase A stimulates Kv7.1 surface expression by regulating Nedd4-2-dependent endocytic trafficking.AMPK: A regulator of ion channels.G-protein-coupled inward rectifier potassium current contributes to ventricular repolarizationA phosphoinositide 3-kinase (PI3K)-serum- and glucocorticoid-inducible kinase 1 (SGK1) pathway promotes Kv7.1 channel surface expression by inhibiting Nedd4-2 protein.Trafficking of the IKs -complex in MDCK cells: site of subunit assembly and determinants of polarized localization.Investigations of the Navβ1b sodium channel subunit in human ventricle; functional characterization of the H162P Brugada syndrome mutant.Genetic variation in the two-pore domain potassium channel, TASK-1, may contribute to an atrial substrate for arrhythmogenesis.
P50
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P50
description
researcher ORCID ID = 0000-0003-1125-1553
@en
wetenschapper
@nl
name
Martin Nybo Andersen
@ast
Martin Nybo Andersen
@en
Martin Nybo Andersen
@es
Martin Nybo Andersen
@nl
type
label
Martin Nybo Andersen
@ast
Martin Nybo Andersen
@en
Martin Nybo Andersen
@es
Martin Nybo Andersen
@nl
prefLabel
Martin Nybo Andersen
@ast
Martin Nybo Andersen
@en
Martin Nybo Andersen
@es
Martin Nybo Andersen
@nl
P106
P31
P496
0000-0003-1125-1553