MiR-216b inhibits pancreatic cancer cell progression and promotes apoptosis by down-regulating

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MiR-216b inhibits pancreatic cancer cell progression and promotes apoptosis by down-regulating

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im Oktober 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована в жовтні 2018

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MiR-216b inhibits pancreatic c ...... s apoptosis by down-regulating

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MiR-216b inhibits pancreatic c ...... s apoptosis by down-regulating

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MiR-216b inhibits pancreatic c ...... s apoptosis by down-regulating

@en

MiR-216b inhibits pancreatic c ...... s apoptosis by down-regulating

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MiR-216b inhibits pancreatic c ...... s apoptosis by down-regulating

@en

MiR-216b inhibits pancreatic c ...... s apoptosis by down-regulating

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P1476

MiR-216b inhibits pancreatic c ...... s apoptosis by down-regulating

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P2093

Baoqiang Wu

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Donglin Sun

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Huihua Cai

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Jun Hu

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Weibo Chen

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Xinquan Wu

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Xuemin Chen

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Yong An

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Yong Jiang

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P2860

Kras as a key oncogene and therapeutic target in pancreatic cancer

The oncogenic kinase Pim-1 is modulated by K-Ras signaling and mediates transformed growth and radioresistance in human pancreatic ductal adenocarcinoma cells.

Inactivation of Ink4a/Arf leads to deregulated expression of miRNAs in K-Ras transgenic mouse model of pancreatic cancer.

Prognostic significance of K-ras mutations in pancreatic cancer: a meta-analysis

miR-216 and miR-217 expression is reduced in transgenic mouse models of pancreatic adenocarcinoma, knockout of miR-216/miR-217 host gene is embryonic lethal.

KRAS mutation is an important predictor of resistance to therapy with epidermal growth factor receptor tyrosine kinase inhibitors in non-small-cell lung cancer.

KRAS Activation and over-expression of SIRT1/BCL6 Contributes to the Pathogenesis of Endometriosis and Progesterone Resistance.

Ect2-Dependent rRNA Synthesis Is Required for KRAS-TRP53-Driven Lung Adenocarcinoma.

MiR-216a decreases MALAT1 expression, induces G2/M arrest and apoptosis in pancreatic cancer cells.

The HIF-2α-MALAT1-miR-216b axis regulates multi-drug resistance of hepatocellular carcinoma cells via modulating autophagy.

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1321-1332

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scholarly article

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10.5114/AOMS.2018.72564

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English

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P478

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2018-10-01T00:00:00Z

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30393486

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6209705

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