E4BP4 promotes thyroid cancer proliferation by modulating iron homeostasis through repression of hepcidin

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E4BP4 promotes thyroid cancer proliferation by modulating iron homeostasis through repression of hepcidin

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im September 2018 veröffentlichter wissenschaftlicher Artikel

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scientific article published on 24 September 2018

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wetenschappelijk artikel

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наукова стаття, опублікована у вересні 2018

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E4BP4 promotes thyroid cancer ...... through repression of hepcidin

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E4BP4 promotes thyroid cancer ...... through repression of hepcidin

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E4BP4 promotes thyroid cancer ...... through repression of hepcidin

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E4BP4 promotes thyroid cancer ...... through repression of hepcidin

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E4BP4 promotes thyroid cancer ...... through repression of hepcidin

@en

E4BP4 promotes thyroid cancer ...... through repression of hepcidin

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P1476

E4BP4 promotes thyroid cancer ...... through repression of hepcidin

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P2093

Jialin Feng

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Jun Chen

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Qinyi Zhou

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P2860

Hepcidin, a urinary antimicrobial peptide synthesized in the liver

Hepcidin regulates cellular iron efflux by binding to ferroportin and inducing its internalization

Protein-protein interaction between the transcriptional repressor E4BP4 and the TBP-binding protein Dr1

G9a histone methyltransferase plays a dominant role in euchromatic histone H3 lysine 9 methylation and is essential for early embryogenesis

Direct interaction between DNMT1 and G9a coordinates DNA and histone methylation during replication

Hepcidin and iron regulation, 10 years later.

Histone lysine methyltransferases as anti-cancer targets for drug discovery

Systemic iron homeostasis and the iron-responsive element/iron-regulatory protein (IRE/IRP) regulatory network

Two to tango: regulation of Mammalian iron metabolism

Ferroportin and iron regulation in breast cancer progression and prognosis

P304

987

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scholarly article

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10.1038/S41419-018-1001-3

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English

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P478

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P50

Jiadong Wang

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2018-09-24T00:00:00Z

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30250199

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P921

immunology

cancer research

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6155336

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