Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

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Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

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im Oktober 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована в жовтні 2018

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Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

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Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

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Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

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Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

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Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

@en

Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

@nl

P1476

Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium

@en

P2093

Alan R Clarke

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Alison Marshall

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Jean C Manson

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P2860

Prion uptake in the gut: identification of the first uptake and replication sites

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Salmonella Transforms Follicle-Associated Epithelial Cells into M Cells to Promote Intestinal Invasion

Botulinum toxin A complex exploits intestinal M cells to enter the host and exert neurotoxicity.

Bacterial colitis increases susceptibility to oral prion disease.

Efficient norovirus and reovirus replication in the mouse intestine requires microfold (M) cells.

Synthetic mammalian prions.

A promoter-level mammalian expression atlas

Variant CJD. 18 years of research and surveillance.

P31

scholarly article

P356

10.1128/JVI.01010-18

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Neil Mabbott

Barry Bradford

P577

2018-10-01T00:00:00Z

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P698

30021891

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P921

immunology

virology

P932

6146811

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