ET receptor activation contributes to T cell accumulation in the kidney following ischemia-reperfusion injury

about

ET receptor activation contributes to T cell accumulation in the kidney following ischemia-reperfusion injury

Item

http://www.wikidata.org/entity/Q58756809

description

im September 2018 veröffentlichter wissenschaftlicher Artikel

@de

scientific article published on 01 September 2018

@en

wetenschappelijk artikel

@nl

наукова стаття, опублікована у вересні 2018

@uk

name

ET receptor activation contrib ...... ng ischemia-reperfusion injury

@en

ET receptor activation contrib ...... ng ischemia-reperfusion injury

@nl

type

Item

label

ET receptor activation contrib ...... ng ischemia-reperfusion injury

@en

ET receptor activation contrib ...... ng ischemia-reperfusion injury

@nl

prefLabel

ET receptor activation contrib ...... ng ischemia-reperfusion injury

@en

ET receptor activation contrib ...... ng ischemia-reperfusion injury

@nl

P1476

ETA receptor activation contri ...... ng ischemia-reperfusion injury

@en

P2093

Erika I Boesen

http://www.w3.org/2001/XMLSchema#string

P2860

Endothelin-1 increases glomerular permeability and inflammation independent of blood pressure in the rat

Lack of an apparent role for endothelin-1 in the prolonged reduction in renal perfusion following severe unilateral ischemia-reperfusion injury in the mouse.

Regulatory T cells suppress innate immunity in kidney ischemia-reperfusion injury

Renal ischemia-reperfusion injury and adenosine 2A receptor-mediated tissue protection: the role of CD4+ T cells and IFN-gamma.

Use of ultrasound to assess renal reperfusion and P-selectin expression following unilateral renal ischemia

Endothelin receptor antagonism prevents hypoxia-induced mortality and morbidity in a mouse model of sickle-cell disease.

Recovery from renal ischemia-reperfusion injury is associated with altered renal hemodynamics, blunted pressure natriuresis, and sodium-sensitive hypertension.

Interleukin 17 promotes angiotensin II-induced hypertension and vascular dysfunction.

Chronic kidney disease after acute kidney injury: a systematic review and meta-analysis

Endothelin-1 is not a Mechanism of IL-17 Induced Hypertension during Pregnancy.

P304

e13865

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.14814/PHY2.13865

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Erika I Boesen

P577

2018-09-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

30198212

http://www.w3.org/2001/XMLSchema#string

P921

reperfusion injury

P932

6129774

http://www.w3.org/2001/XMLSchema#string