Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

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Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

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im Januar 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

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Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

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Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

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Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

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Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

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Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

@nl

P1476

Suppression of c-MET overcomes erlotinib resistance in tongue cancer cells

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Dongxu Liu

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Keqiang Huang

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P2860

MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling.

EGFR Tyrosine 845 Phosphorylation-Dependent Proliferation and Transformation of Breast Cancer Cells Require Activation of p38 MAPK.

The quest to overcome resistance to EGFR-targeted therapies in cancer.

Tongue carcinoma infrequently harbor common actionable genetic alterations

Targeting epidermal growth factor receptor in head and neck cancer.

MET FISH-positive status predicts short progression-free survival and overall survival after gefitinib treatment in lung adenocarcinoma with EGFR mutation

A recombinant decoy comprising EGFR and ErbB-4 inhibits tumor growth and metastasis.

Prognostic value of MET, cyclin D1 and MET gene copy number in non-small cell lung cancer

Dysregulated molecular networks in head and neck carcinogenesis

Multidrug resistance in oral squamous cell carcinoma: The role of vacuolar ATPases.

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5499-5508

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P31

scholarly article

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10.2147/OTT.S167936

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English

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2018-01-01T00:00:00Z

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30233210

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6134955

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