Mucosal-Associated Invariant T Cells Improve Nonalcoholic Fatty Liver Disease Through Regulating Macrophage Polarization

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Mucosal-Associated Invariant T Cells Improve Nonalcoholic Fatty Liver Disease Through Regulating Macrophage Polarization

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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Mucosal-Associated Invariant T ...... lating Macrophage Polarization

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Mucosal-Associated Invariant T ...... lating Macrophage Polarization

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Mucosal-Associated Invariant T ...... lating Macrophage Polarization

@en

Mucosal-Associated Invariant T ...... lating Macrophage Polarization

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Mucosal-Associated Invariant T ...... lating Macrophage Polarization

@en

Mucosal-Associated Invariant T ...... lating Macrophage Polarization

@nl

P1476

Mucosal-Associated Invariant T ...... lating Macrophage Polarization

@en

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Bingyuan Huang

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Jingyuan Fang

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Jun Zhang

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Min Lian

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Qi Miao

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Qixia Wang

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Ruqi Tang

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Weihua Chen

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Xiang Jiang

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Xiong Ma

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The Immune Landscape in Nonalcoholic Steatohepatitis.

Mucosal-associated invariant T cells in autoimmunity, immune-mediated diseases and airways disease

MR1-Restricted Mucosal-Associated Invariant T Cells and Their Activation during Infectious Diseases

MR1 presents microbial vitamin B metabolites to MAIT cells

T-cell activation by transitory neo-antigens derived from distinct microbial pathways

MAIT cells: new guardians of the liver

TLR4 links innate immunity and fatty acid-induced insulin resistance

Programmed cell death 1 forms negative costimulatory microclusters that directly inhibit T cell receptor signaling by recruiting phosphatase SHP2.

The identification of markers of macrophage differentiation in PMA-stimulated THP-1 cells and monocyte-derived macrophages.

Depletion of liver Kupffer cells prevents the development of diet-induced hepatic steatosis and insulin resistance.

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1994

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scholarly article

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10.3389/FIMMU.2018.01994

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English

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M. Eric Gershwin

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2018-01-01T00:00:00Z

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30233587

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macrophage

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6131560

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