HJURP promotes hepatocellular carcinoma proliferation by destabilizing p21 via the MAPK/ERK1/2 and AKT/GSK3β signaling pathways

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HJURP promotes hepatocellular carcinoma proliferation by destabilizing p21 via the MAPK/ERK1/2 and AKT/GSK3β signaling pathways

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im August 2018 veröffentlicher wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована в серпні 2018

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HJURP promotes hepatocellular ...... d AKT/GSK3β signaling pathways

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HJURP promotes hepatocellular ...... d AKT/GSK3β signaling pathways

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HJURP promotes hepatocellular ...... d AKT/GSK3β signaling pathways

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HJURP promotes hepatocellular ...... d AKT/GSK3β signaling pathways

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HJURP promotes hepatocellular ...... d AKT/GSK3β signaling pathways

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HJURP promotes hepatocellular ...... d AKT/GSK3β signaling pathways

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P1476

HJURP promotes hepatocellular ...... d AKT/GSK3β signaling pathways

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Hechen Huang

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Lei Geng

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Lin Zhou

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Shengyong Yin

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Shusen Zheng

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Tian Shen

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Tianchi Chen

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Yuan Zhou

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Selective ubiquitylation of p21 and Cdt1 by UBCH8 and UBE2G ubiquitin-conjugating enzymes via the CRL4Cdt2 ubiquitin ligase complex.

PCNA-dependent regulation of p21 ubiquitylation and degradation via the CRL4Cdt2 ubiquitin ligase complex.

HJURP is a cell-cycle-dependent maintenance and deposition factor of CENP-A at centromeres

Global cancer statistics, 2012

Efficacy and safety of sorafenib in patients in the Asia-Pacific region with advanced hepatocellular carcinoma: a phase III randomised, double-blind, placebo-controlled trial

Modulation of HJURP (Holliday Junction-Recognizing Protein) levels is correlated with glioblastoma cells survival

The expression level of HJURP has an independent prognostic impact and predicts the sensitivity to radiotherapy in breast cancer.

XAF1 directs apoptotic switch of p53 signaling through activation of HIPK2 and ZNF313

KLF4, p21 and context-dependent opposing forces in cancer.

CRL2(LRR-1) targets a CDK inhibitor for cell cycle control in C. elegans and actin-based motility regulation in human cells

P2888

s13046-018-0866-4

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scholarly article

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10.1186/S13046-018-0866-4

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P478

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2018-08-15T00:00:00Z

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1106151496

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30111352

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6094877

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