Maf1 and Repression of RNA Polymerase III-Mediated Transcription Drive Adipocyte Differentiation

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Maf1 and Repression of RNA Polymerase III-Mediated Transcription Drive Adipocyte Differentiation

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im August 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована в серпні 2018

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Maf1 and Repression of RNA Pol ...... rive Adipocyte Differentiation

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Maf1 and Repression of RNA Pol ...... rive Adipocyte Differentiation

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Maf1 and Repression of RNA Pol ...... rive Adipocyte Differentiation

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Maf1 and Repression of RNA Pol ...... rive Adipocyte Differentiation

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Maf1 and Repression of RNA Pol ...... rive Adipocyte Differentiation

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Maf1 and Repression of RNA Pol ...... rive Adipocyte Differentiation

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Maf1 and Repression of RNA Pol ...... rive Adipocyte Differentiation

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Christopher J Walkey

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Chun-Yuan Chen

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Deborah L Johnson

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Rainer B Lanz

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Wange Lu

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Wen-Hsuan Chang

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P2860

The product of the H19 gene may function as an RNA

Transcription by RNA polymerase III: insights into mechanism and regulation

Enhanced RNA polymerase III-dependent transcription is required for oncogenic transformation

ALKBH1-Mediated tRNA Demethylation Regulates Translation

Adipocyte differentiation from the inside out

Linkage study and exome sequencing identify a BDP1 mutation associated with hereditary hearing loss

Mutations in genes encoding subunits of RNA polymerases I and III cause Treacher Collins syndrome.

Maf1 is an essential mediator of diverse signals that repress RNA polymerase III transcription.

A novel role for an RNA polymerase III subunit POLR3G in regulating pluripotency in human embryonic stem cells.

Mammalian Maf1 is a negative regulator of transcription by all three nuclear RNA polymerases.

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1852-1864

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scholarly article

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10.1016/J.CELREP.2018.07.046

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English

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2018-08-14T00:00:00Z

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30110641

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6138453

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