Adhesion of Staphylococcus aureus to Corneocytes from Atopic Dermatitis Patients Is Controlled by Natural Moisturizing Factor Levels

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Adhesion of Staphylococcus aureus to Corneocytes from Atopic Dermatitis Patients Is Controlled by Natural Moisturizing Factor Levels

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scientific article published on 14 August 2018

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wetenschappelijk artikel

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наукова стаття, опублікована в серпні 2018

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Adhesion of Staphylococcus aur ...... ral Moisturizing Factor Levels

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Adhesion of Staphylococcus aur ...... ral Moisturizing Factor Levels

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Adhesion of Staphylococcus aur ...... ral Moisturizing Factor Levels

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Adhesion of Staphylococcus aur ...... ral Moisturizing Factor Levels

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Adhesion of Staphylococcus aur ...... ral Moisturizing Factor Levels

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Adhesion of Staphylococcus aur ...... ral Moisturizing Factor Levels

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P1476

Adhesion of Staphylococcus aur ...... ral Moisturizing Factor Levels

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Cécile Feuillie

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Maeve A McAleer

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Pauline Vitry

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Sanja Kezic

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P2860

Physical stress and bacterial colonization

A "dock, lock, and latch" structural model for a staphylococcal adhesin binding to fibrinogen

Evidence for the "dock, lock, and latch" ligand binding mechanism of the staphylococcal microbial surface component recognizing adhesive matrix molecules (MSCRAMM) SdrG

Crystal Structures Reveal the Multi-Ligand Binding Mechanism of Staphylococcus aureus ClfB

The mechanical world of bacteria

Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis

The Burden of Atopic Dermatitis: Summary of a Report for the National Eczema Association.

Roles for fibrinogen, immunoglobulin and complement in platelet activation promoted by Staphylococcus aureus clumping factor A.

Filaggrin mutations associated with skin and allergic diseases.

Loss-of-function mutations in the gene encoding filaggrin cause ichthyosis vulgaris.

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scholarly article

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10.1128/MBIO.01184-18

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English

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Yves F Dufrêne

Alan D. Irvine

Joan A Geoghegan

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2018-08-14T00:00:00Z

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30108169

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6094479

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