about
Mycobacterium tuberculosis responds to chloride and pH as synergistic cues to the immune status of its host cellStrategies used by bacterial pathogens to suppress plant defensesNovel inhibitors of cholesterol degradation in Mycobacterium tuberculosis reveal how the bacterium's metabolism is constrained by the intracellular environmentBacterial elicitation and evasion of plant innate immunity.Genomewide identification of Pseudomonas syringae pv. tomato DC3000 promoters controlled by the HrpL alternative sigma factor.Mycobacterium tuberculosis wears what it eatsSlow growth of Mycobacterium tuberculosis at acidic pH is regulated by phoPR and host-associated carbon sources.Pseudomonas type III effector AvrPtoB induces plant disease susceptibility by inhibition of host programmed cell death.A bacterial inhibitor of host programmed cell death defenses is an E3 ubiquitin ligase.Type III effector AvrPtoB requires intrinsic E3 ubiquitin ligase activity to suppress plant cell death and immunity.The Carbonic Anhydrase Inhibitor Ethoxzolamide Inhibits the Mycobacterium tuberculosis PhoPR Regulon and Esx-1 Secretion and Attenuates Virulence.SPARTA: Simple Program for Automated reference-based bacterial RNA-seq Transcriptome AnalysisAvrPtoB: a bacterial type III effector that both elicits and suppresses programmed cell death associated with plant immunity.Inhibitors of Mycobacterium tuberculosis DosRST signaling and persistence.The N-terminal region of Pseudomonas type III effector AvrPtoB elicits Pto-dependent immunity and has two distinct virulence determinants.Immune activation of the host cell induces drug tolerance in Mycobacterium tuberculosis both in vitro and in vivo.A Nonsense Mutation in Mycobacterium marinum That Is Suppressible by a Novel MechanismPolysorbates prevent biofilm formation and pathogenesis of Escherichia coli O104:H4.Small Molecules That Sabotage Bacterial Virulence.A bioluminescent Pseudomonas aeruginosa wound model reveals increased mortality of type 1 diabetic mice to biofilm infection.2-aminoimidazoles potentiate ß-lactam antimicrobial activity against Mycobacterium tuberculosis by reducing ß-lactamase secretion and increasing cell envelope permeability.Macrophage infection models for Mycobacterium tuberculosis.aprABC: a Mycobacterium tuberculosis complex-specific locus that modulates pH-driven adaptation to the macrophage phagosome.Diverse AvrPtoB homologs from several Pseudomonas syringae pathovars elicit Pto-dependent resistance and have similar virulence activities.A bacterial E3 ubiquitin ligase targets a host protein kinase to disrupt plant immunity.The ukb1 gene encodes a putative protein kinase required for bud site selection and pathogenicity in Ustilago maydis.WhiB6 regulation of ESX-1 gene expression is controlled by a negative feedback loop in Mycobacterium marinum.Targeting Mycobacterium tuberculosis Sensitivity to Thiol Stress at Acidic pH Kills the Bacterium and Potentiates Antibiotics.HC2091 kills Mycobacterium tuberculosis by targeting the MmpL3 mycolic acid transporter.Genetic and metabolic regulation of Mycobacterium tuberculosis acid growth arrest.Mycobacterium tuberculosis Invasion of Macrophages: Linking Bacterial Gene Expression to Environmental CuesRemodeling of Mycobacterium tuberculosis lipids regulates prpCD during acid growth arrest2-aminoimidazoles collapse mycobacterial proton motive force and block the electron transport chainIdentification of new MmpL3 inhibitors by untargeted and targeted mutant screens defines MmpL3 domains with differential resistanceMultiple-stage Precursor Ion Separation and High Resolution Mass Spectrometry toward Structural Characterization of 2,3-Diacyltrehalose Family from Mycobacterium tuberculosisMycobacterium tuberculosis Reporter Strains as Tools for Drug Discovery and DevelopmentInhibition of Mycobacterium tuberculosis DosRST two-component regulatory system signaling by targeting response regulator DNA binding and sensor kinase hemeGenetic and metabolic regulation ofMycobacterium tuberculosisacid growth arrestAcidic pH-dependent depletion of Mycobacterium tuberculosis thiol pools potentiates antibiotics and oxidizing agentsEspM Is a Conserved Transcription Factor That Regulates Gene Expression in Response to the ESX-1 System
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P50
description
researcher ORCID ID = 0000-0002-4119-4169
@en
wetenschapper
@nl
name
Robert B Abramovitch
@ast
Robert B Abramovitch
@en
Robert B Abramovitch
@es
Robert B Abramovitch
@nl
type
label
Robert B Abramovitch
@ast
Robert B Abramovitch
@en
Robert B Abramovitch
@es
Robert B Abramovitch
@nl
prefLabel
Robert B Abramovitch
@ast
Robert B Abramovitch
@en
Robert B Abramovitch
@es
Robert B Abramovitch
@nl
P106
P1153
57200132052
7006021365
P21
P31
P496
0000-0002-4119-4169