CD40L/IL-4-stimulated CLL demonstrates variation in translational regulation of DNA damage response genes including ATM

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CD40L/IL-4-stimulated CLL demonstrates variation in translational regulation of DNA damage response genes including ATM

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wetenschappelijk artikel

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наукова стаття, опублікована в серпні 2018

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CD40L/IL-4-stimulated CLL demo ...... e response genes including ATM

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CD40L/IL-4-stimulated CLL demo ...... e response genes including ATM

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CD40L/IL-4-stimulated CLL demo ...... e response genes including ATM

@en

CD40L/IL-4-stimulated CLL demo ...... e response genes including ATM

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CD40L/IL-4-stimulated CLL demo ...... e response genes including ATM

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CD40L/IL-4-stimulated CLL demo ...... e response genes including ATM

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P1476

CD40L/IL-4-stimulated CLL demo ...... e response genes including ATM

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P2093

Aleksandra Bzura

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Anne E Willis

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Carolyn Jones

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Daniel Beck

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George D D Jones

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Kate M Dudek

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Larissa Lezina

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Ruth V Spriggs

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P2860

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

Functional interaction of H2AX, NBS1, and p53 in ATM-dependent DNA damage responses and tumor suppression

Guidelines for the diagnosis and treatment of chronic lymphocytic leukemia: a report from the International Workshop on Chronic Lymphocytic Leukemia updating the National Cancer Institute-Working Group 1996 guidelines

Genome-wide analysis in vivo of translation with nucleotide resolution using ribosome profiling

Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources

Bioinformatics enrichment tools: paths toward the comprehensive functional analysis of large gene lists

ATM phosphorylates histone H2AX in response to DNA double-strand breaks

Ataxia-telangiectasia: structural diversity of untranslated sequences suggests complex post-transcriptional regulation of ATM gene expression

Genome-wide assessment of differential translations with ribosome profiling data.

Translational reprogramming following UVB irradiation is mediated by DNA-PKcs and allows selective recruitment to the polysomes of mRNAs encoding DNA repair enzymes.

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1869-1881

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scholarly article

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10.1182/BLOODADVANCES.2017015560

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English

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P478

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Simon D Wagner

Graham Packham

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2018-08-01T00:00:00Z

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30082430

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6093746

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