Chronic pro-oxidative state and mitochondrial dysfunctions are more pronounced in fibroblasts from Down syndrome foeti with congenital heart defects
about
Genomic instability: Crossing pathways at the origin of structural and numerical chromosome changes.Overexpression of Chromosome 21 miRNAs May Affect Mitochondrial Function in the Hearts of Down Syndrome Fetuses.Metabolic and Growth Rate Alterations in Lymphoblastic Cell Lines Discriminate Between Down Syndrome and Alzheimer's Disease.
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Chronic pro-oxidative state and mitochondrial dysfunctions are more pronounced in fibroblasts from Down syndrome foeti with congenital heart defects
description
im Dezember 2012 veröffentlichter wissenschaftlicher Artikel
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scientific article published on 20 December 2012
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wetenschappelijk artikel
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наукова стаття, опублікована в грудні 2012
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name
Chronic pro-oxidative state an ...... with congenital heart defects
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Chronic pro-oxidative state an ...... with congenital heart defects
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type
label
Chronic pro-oxidative state an ...... with congenital heart defects
@en
Chronic pro-oxidative state an ...... with congenital heart defects
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prefLabel
Chronic pro-oxidative state an ...... with congenital heart defects
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Chronic pro-oxidative state an ...... with congenital heart defects
@nl
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Chronic pro-oxidative state an ...... with congenital heart defects
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Claudia Piccoli
Flaviana Gentile
Gaetano Calì
Lucio Nitsch
Marina Prisco
Nazzareno Capitanio
Rosa Negri
Rosanna Manco
Rosella Scrima
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P304
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10.1093/HMG/DDS529
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2012-12-20T00:00:00Z