Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy
about
The NO/ONOO-cycle as the central cause of heart failure.Dantrolene improves in vitro structural changes induced by serum from Trypanosoma cruzi-infected mice.Increased mitochondrial emission of reactive oxygen species and calpain activation are required for doxorubicin-induced cardiac and skeletal muscle myopathy.Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure.
P2860
Calpain-mediated dystrophin disruption may be a potential structural culprit behind chronic doxorubicin-induced cardiomyopathy
description
article
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im November 2011 veröffentlichter wissenschaftlicher Artikel
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wetenschappelijk artikel
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наукова стаття, опублікована в листопаді 2011
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name
Calpain-mediated dystrophin di ...... rubicin-induced cardiomyopathy
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Calpain-mediated dystrophin di ...... rubicin-induced cardiomyopathy
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type
label
Calpain-mediated dystrophin di ...... rubicin-induced cardiomyopathy
@en
Calpain-mediated dystrophin di ...... rubicin-induced cardiomyopathy
@nl
prefLabel
Calpain-mediated dystrophin di ...... rubicin-induced cardiomyopathy
@en
Calpain-mediated dystrophin di ...... rubicin-induced cardiomyopathy
@nl
P2093
P50
P1476
Calpain-mediated dystrophin di ...... rubicin-induced cardiomyopathy
@en
P2093
Erica C Campos
João L O'Connell
Lygia M Malvestio
Marcos A Rossi
Marcus V Simões
Simone G Ramos
P304
P356
10.1016/J.EJPHAR.2011.09.021
P407
P577
2011-09-21T00:00:00Z