about
Lesion of the subiculum reduces the spread of amyloid beta pathology to interconnected brain regions in a mouse model of Alzheimer's diseaseIdentification and description of three families with familial Alzheimer disease that segregate variants in the SORL1 gene.Autophagic and lysosomal defects in human tauopathies: analysis of post-mortem brain from patients with familial Alzheimer disease, corticobasal degeneration and progressive supranuclear palsy.Modulation of the endoplasmic reticulum-mitochondria interface in Alzheimer's disease and related models.Aggregation of the Inflammatory S100A8 Precedes Aβ Plaque Formation in Transgenic APP Mice: Positive Feedback for S100A8 and Aβ Productions.Increased Epileptiform EEG Activity and Decreased Seizure Threshold in Arctic APP Transgenic Mouse Model of Alzheimer's Disease.Diurnal effects of enriched environment on immediate early gene expression in the rat brain.Effects of postischemic environment on transcription factor and serotonin receptor expression after permanent focal cortical ischemia in rats.Neuropathological characterization of two siblings carrying the MAPT S305S mutation demonstrates features resembling argyrophilic grain disease.Mitochondrial dysfunction in a transgenic mouse model expressing human amyloid precursor protein (APP) with the Arctic mutation.Environmental enrichment reverses learning impairment in the Morris water maze after focal cerebral ischemia in rats.Enriched environment increases spinophilin mRNA expression and spinophilin immunoreactive dendritic spines in hippocampus and cortex.Progressive neuropathology and cognitive decline in a single Arctic APP transgenic mouse model.Critical role of somatostatin receptor 2 in the vulnerability of the central noradrenergic system: new aspects on Alzheimer's disease.Amyloid precursor protein accumulates in aggresomes in response to proteasome inhibitor.Gene expression profiling of the rat hippocampus one month after focal cerebral ischemia followed by enriched environment.Amyloid neuropathology in the single Arctic APP transgenic model affects interconnected brain regions.β-amyloid(25-35) inhibits the activity of inositol (1,4,5)-trisphosphate-5-phosphataseGlucocorticoid axis abnormalities related to brain damage
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description
onderzoeker
@nl
researcher ORCID ID = 0000-0001-5561-0197
@en
name
Annica Rönnbäck
@ast
Annica Rönnbäck
@en
Annica Rönnbäck
@nl
type
label
Annica Rönnbäck
@ast
Annica Rönnbäck
@en
Annica Rönnbäck
@nl
prefLabel
Annica Rönnbäck
@ast
Annica Rönnbäck
@en
Annica Rönnbäck
@nl
P106
P31
P496
0000-0001-5561-0197