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Gap junctions support the sustained phase of hypoxic pulmonary vasoconstriction by facilitating calcium sensitization.Protein kinase C in enhanced vascular tone in diabetes mellitus.Rho kinase and protein kinase C involvement in vascular smooth muscle myofilament calcium sensitization in arteries from diabetic rats.Electrophysiological and contractile evidence of the ability of human mesenchymal stromal cells to correct vascular malfunction in rats after ionizing irradiation.Arrhythmogenic peroxynitrite-induced alterations in mammalian heart contractility and its prevention with quercetin-filled liposomes.Evidence for the role of phosphatidylcholine-specific phospholipase C in sustained hypoxic pulmonary vasoconstriction.Nitric oxide relaxes rat tail artery smooth muscle by cyclic GMP-independent decrease in calcium sensitivity of myofilaments.Selective glycolysis blockade in guinea pig pulmonary artery and aorta reverses contractile and electrical responses to acute hypoxia.Quercetin-filled phosphatidylcholine liposomes restore abnormalities in rat thoracic aorta BK(Ca) channel function following ionizing irradiation.Functional and molecular consequences of ionizing irradiation on large conductance Ca2+-activated K+ channels in rat aortic smooth muscle cells.PKC-δ isozyme gene silencing restores vascular function in diabetic rat.Correction of vascular hypercontractility in spontaneously hypertensive rats using shRNAs-induced delta protein kinase C gene silencing.Phospholipid vesicles (liposomes) restore endothelium-dependent cholinergic relaxation in thoracic aorta from spontaneously hypertensive rats.The contractile apparatus in vascular smooth muscle cells of spontaneously hypertensive rats possess increased calcium sensitivity: the possible role of protein kinase CEvidence for decrease in myofilament responsiveness to Ca2+ during hypoxia in spontaneously active vascular smooth muscle in ratsThe similarity in action of hypoxia and platelet-activating factor on smooth muscle cells of coronary arteries: possible explanation for hypoxic coronary spasm developmentEvidence for the involvement of protein kinase C in depression of endothelium-dependent vascular responses in spontaneously hypertensive ratsIonizing non-fatal whole-body irradiation inhibits Ca2+-dependent K+ channels in endothelial cells of rat coronary artery: possible contribution to depression of endothelium-dependent vascular relaxationHypoxic pulmonary vasoconstriction is lacking in rats with type 1 diabetes
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description
researcher ORCID ID = 0000-0003-0976-2792
@en
name
Anatoly Soloviev
@ast
Anatoly Soloviev
@en
Anatoly Soloviev
@es
Anatoly Soloviev
@nl
type
label
Anatoly Soloviev
@ast
Anatoly Soloviev
@en
Anatoly Soloviev
@es
Anatoly Soloviev
@nl
prefLabel
Anatoly Soloviev
@ast
Anatoly Soloviev
@en
Anatoly Soloviev
@es
Anatoly Soloviev
@nl
P106
P31
P496
0000-0003-0976-2792