Ingenol mebutate induces a tumor cell-directed inflammatory response and antimicrobial peptides thereby promoting rapid tumor destruction and wound healing

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Ingenol mebutate induces a tumor cell-directed inflammatory response and antimicrobial peptides thereby promoting rapid tumor destruction and wound healing

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im September 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована у вересні 2018

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Ingenol mebutate induces a tum ...... destruction and wound healing

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Ingenol mebutate induces a tum ...... destruction and wound healing

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Ingenol mebutate induces a tum ...... destruction and wound healing

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Ingenol mebutate induces a tum ...... destruction and wound healing

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Ingenol mebutate induces a tum ...... destruction and wound healing

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Ingenol mebutate induces a tum ...... destruction and wound healing

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P1476

Ingenol mebutate induces a tum ...... destruction and wound healing

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Bernhard Homey

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Bettina Alexandra Buhren

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Edwin Bölke

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Holger Schrumpf

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Julia Baran

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Peter Arne Gerber

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Stephan Alexander Braun

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P2860

Isolation and characterization of human beta -defensin-3, a novel human inducible peptide antibiotic

RNase 7, a novel innate immune defense antimicrobial protein of healthy human skin

Cutting edge: the orphan chemokine receptor G protein-coupled receptor-2 (GPR-2, CCR10) binds the skin-associated chemokine CCL27 (CTACK/ALP/ILC)

Monocyte chemoattractant protein 1 acts as a T-lymphocyte chemoattractant

Imiquimod: mode of action

Systematic identification and characterization of novel human skin-associated genes encoding membrane and secreted proteins

Human antimicrobial peptides and proteins

The skin cancer chemotherapeutic agent ingenol-3-angelate (PEP005) is a substrate for the epidermal multidrug transporter (ABCB1) and targets tumor vasculature.

Dual mechanism of action of ingenol mebutate gel for topical treatment of actinic keratoses: rapid lesion necrosis followed by lesion-specific immune response.

Ingenol mebutate gel for actinic keratosis.

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s40001-018-0343-8

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scholarly article

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10.1186/S40001-018-0343-8

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English

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P478

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P577

2018-09-28T00:00:00Z

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30266096

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P932

6161468

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