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The Na+-dependent chloride-bicarbonate exchanger SLC4A8 mediates an electroneutral Na+ reabsorption process in the renal cortical collecting ducts of miceContractile force is enhanced in Aortas from pendrin null mice due to stimulation of angiotensin II-dependent signaling.Pendrin gene ablation alters ENaC subcellular distribution and open probabilityENaC inhibition stimulates HCl secretion in the mouse cortical collecting duct. II. Bafilomycin-sensitive H+ secretionENaC inhibition stimulates HCl secretion in the mouse cortical collecting duct. I. Stilbene-sensitive Cl- secretion.Pendrin protein abundance in the kidney is regulated by nitric oxide and cAMPAngiotensin II activates H+-ATPase in type A intercalated cells.Nitric oxide reduces Cl⁻ absorption in the mouse cortical collecting duct through an ENaC-dependent mechanism.The interaction of pendrin and the epithelial sodium channel in blood pressure regulation.Epac regulates UT-A1 to increase urea transport in inner medullary collecting ducts.Blatchford Score Is Superior to AIMS65 Score in Predicting the Need for Clinical Interventions in Elderly Patients with Nonvariceal Upper Gastrointestinal Bleed.Vasopressin increases urea permeability in the initial IMCD from diabetic rats.Angiotensin II increases chloride absorption in the cortical collecting duct in mice through a pendrin-dependent mechanismOxidant stress and blood pressure responses to angiotensin II administration in rats fed varying salt dietsThe Na-dependent chloride-bicarbonate exchanger SLC4A8 mediates an electroneutral Na reabsorption process in the renal cortical collecting ducts of mice
P50
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P50
description
researcher ORCID ID = 0000-0002-1994-4277
@en
wetenschapper
@nl
name
Vladimir Pech
@ast
Vladimir Pech
@en
Vladimir Pech
@es
Vladimir Pech
@nl
type
label
Vladimir Pech
@ast
Vladimir Pech
@en
Vladimir Pech
@es
Vladimir Pech
@nl
prefLabel
Vladimir Pech
@ast
Vladimir Pech
@en
Vladimir Pech
@es
Vladimir Pech
@nl
P31
P496
0000-0002-1994-4277