F 16915 prevents heart failure-induced atrial fibrillation: a promising new drug as upstream therapy

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Omega-3 Index and Anti-Arrhythmic Potential of Omega-3 PUFAs.DL-3-n-Butylphthalide reduces atrial fibrillation susceptibility by inhibiting atrial structural remodeling in rats with heart failure.

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F 16915 prevents heart failure-induced atrial fibrillation: a promising new drug as upstream therapy

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http://www.wikidata.org/entity/Q59248658

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im April 2014 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована у квітні 2014

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F 16915 prevents heart failure ...... g new drug as upstream therapy

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F 16915 prevents heart failure ...... g new drug as upstream therapy

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F 16915 prevents heart failure ...... g new drug as upstream therapy

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F 16915 prevents heart failure ...... g new drug as upstream therapy

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F 16915 prevents heart failure ...... g new drug as upstream therapy

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F 16915 prevents heart failure ...... g new drug as upstream therapy

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F 16915 prevents heart failure ...... g new drug as upstream therapy

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Bruno Le Grand

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Elisabeth Dupont-Passelaigue

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Frédéric Longo

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Frédérique Lantoine-Adam

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Georghia Michael

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Kunihiro Nishida

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Philippe Legrand

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Robert Letienne

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Differential efficacy of L- and T-type calcium channel blockers in preventing tachycardia-induced atrial remodeling in dogs.

Systems approach with inflammatory exudates uncovers novel anti-inflammatory and pro-resolving mediators.

Effects of dietary omega-3 fatty acids on ventricular function in dogs with healed myocardial infarctions: in vivo and in vitro studies

Treatment with docosahexaenoic acid, but not eicosapentaenoic acid, delays Ca2+-induced mitochondria permeability transition in normal and hypertrophied myocardium.

Omega-3 fatty acids and cardiovascular disease: effects on risk factors, molecular pathways, and clinical events.

New ideas about atrial fibrillation 50 years on.

Direct protective effects of poly-unsaturated fatty acids, DHA and EPA, against activation of cardiac late sodium current: a mechanism for ischemia selectivity.

Regulation of connexin biosynthesis, assembly, gap junction formation, and removal.

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s00210-014-0975-3

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667-677

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scholarly article

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10.1007/S00210-014-0975-3

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387

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Stanley Nattel

Stéphane Hatem

Monique David-Dufilho

Daniel Catheline

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2014-04-11T00:00:00Z

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24722816

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F 16915 prevents heart failure-induced atrial fibrillation: a promising new drug as upstream therapy

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F 16915 prevents heart failure-induced atrial fibrillation: a promising new drug as upstream therapy

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