about
Loss and recovery of Mgat3 and GnT-III Mediated E-cadherin N-glycosylation is a mechanism involved in epithelial-mesenchymal-epithelial transitions.Insulin/IGF-I signaling pathways enhances tumor cell invasion through bisecting GlcNAc N-glycans modulation. an interplay with E-cadherin.E-cadherin and adherens-junctions stability in gastric carcinoma: functional implications of glycosyltransferases involving N-glycan branching biosynthesis, N-acetylglucosaminyltransferases III and V.Dysregulation of T cell receptor N-glycosylation: a molecular mechanism involved in ulcerative colitis.Macrophages from IBD patients exhibit defective tumour necrosis factor-α secretion but otherwise normal or augmented pro-inflammatory responses to infection.Editorial: A stake in the game: ADAM23 contributes to dendritic cell effectiveness in stimulating CD4+ T cell proliferative responses
P50
description
researcher ORCID ID = 0000-0002-3496-5463
@en
name
Joana Cabral
@ast
Joana Cabral
@en
Joana Cabral
@es
Joana Cabral
@nl
type
label
Joana Cabral
@ast
Joana Cabral
@en
Joana Cabral
@es
Joana Cabral
@nl
prefLabel
Joana Cabral
@ast
Joana Cabral
@en
Joana Cabral
@es
Joana Cabral
@nl
P1153
57191837489
P21
P31
P496
0000-0002-3496-5463