about
Prenatal cerebral ischemia triggers dysmaturation of caudate projection neurons.Role of recurrent hypoxia-ischemia in preterm white matter injury severity.Characterization of Ribeye subunits in zebrafish hair cells reveals that exogenous Ribeye B-domain and CtBP1 localize to the basal ends of synaptic ribbons.Prenatal cerebral ischemia disrupts MRI-defined cortical microstructure through disturbances in neuronal arborization.Thrombogenicity of flow diverters in an ex vivo shunt model: effect of phosphorylcholine surface modification.A TLR/AKT/FoxO3 immune tolerance-like pathway disrupts the repair capacity of oligodendrocyte progenitors.Static spatial growth restriction micropatterning of endothelial colony forming cells influences their morphology and gene expression.Luminal Plasma Treatment for Small Diameter Polyvinyl Alcohol Tubular Scaffolds.Biomimetic modification of poly(vinyl alcohol): Encouraging endothelialization and preventing thrombosis with antiplatelet monotherapyContact Activation Inhibitor and Factor XI Antibody, AB023, Produces Safe, Dose-Dependent Anticoagulation in a Phase 1 First-In-Human TrialTransient Hypoxemia Disrupts Anatomical and Functional Maturation of Preterm Fetal Ovine CA1 Pyramidal NeuronsEndothelial PAI-1 (Plasminogen Activator Inhibitor-1) Blocks the Intrinsic Pathway of Coagulation, Inducing the Clearance and Degradation of FXIa (Activated Factor XI)
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description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-9812-2262
@en
name
Matthew W Hagen
@ast
Matthew W Hagen
@en
Matthew W Hagen
@nl
type
label
Matthew W Hagen
@ast
Matthew W Hagen
@en
Matthew W Hagen
@nl
prefLabel
Matthew W Hagen
@ast
Matthew W Hagen
@en
Matthew W Hagen
@nl
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P106
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0000-0002-9812-2262