about
The sst1 resistance locus regulates evasion of type I interferon signaling by Chlamydia pneumoniae as a disease tolerance mechanismCD14 cooperates with complement receptor 3 to mediate MyD88-independent phagocytosis of Borrelia burgdorferiMouse strain-dependent differences in susceptibility to Neisseria gonorrhoeae infection and induction of innate immune responses.Fibroblast growth factor-inducible 14 (Fn14) is expressed in the lower genital tract and may play a role in amplifying inflammation during infection.Lipopolysaccharide recognition, CD14, and lipopolysaccharide receptors.Enhanced virulence of Chlamydia muridarum respiratory infections in the absence of TLR2 activation.Membrane-associated proteins of a lipopolysaccharide-deficient mutant of Neisseria meningitidis activate the inflammatory response through toll-like receptor 2.Plasmid-cured Chlamydia caviae activates TLR2-dependent signaling and retains virulence in the guinea pig model of genital tract infection.The inflammatory cytokine response to Chlamydia trachomatis infection is endotoxin mediated.Protective role of Toll-like receptor 4 in experimental gonococcal infection of female miceComparative analysis of the growth and biological activity of a respiratory and atheroma isolate of Chlamydia pneumoniae reveals strain-dependent differences in inflammatory activity and innate immune evasion.CD11c/CD18, a transmembrane signaling receptor for lipopolysaccharideLocalization of TLR2 and MyD88 to Chlamydia trachomatis inclusions. Evidence for signaling by intracellular TLR2 during infection with an obligate intracellular pathogen.Strongyloides stercoralis transmission by kidney transplantation in two recipients from a common donor.The Lip lipoprotein from Neisseria gonorrhoeae stimulates cytokine release and NF-kappaB activation in epithelial cells in a Toll-like receptor 2-dependent manner.Induction of tolerance to lipopolysaccharide and mycobacterial components in Chinese hamster ovary/CD14 cells is not affected by overexpression of Toll-like receptors 2 or 4.Divergent response to LPS and bacteria in CD14-deficient murine macrophages.Membrane expression of soluble endotoxin-binding proteins permits lipopolysaccharide signaling in Chinese hamster ovary fibroblasts independently of CD14.Cellular Activation, Phagocytosis, and Bactericidal Activity Against Group B Streptococcus Involve Parallel Myeloid Differentiation Factor 88-Dependent and Independent Signaling PathwaysCD11/CD18 leukocyte integrins: new signaling receptors for bacterial endotoxinDifferential roles of TLR2 and TLR4 in the host response to Gram-negative bacteria: lessons from a lipopolysaccharide-deficient mutant of Neisseria meningitidisInvolvement of CD14 and complement receptors CR3 and CR4 in nuclear factor-kappaB activation and TNF production induced by lipopolysaccharide and group B streptococcal cell wallsThe CD11/CD18 integrins: characterization of three novel LPS signaling receptors
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description
researcher ORCID ID = 0000-0003-2197-7198
@en
wetenschapper
@nl
name
Robin R Ingalls
@ast
Robin R Ingalls
@en
Robin R Ingalls
@nl
type
label
Robin R Ingalls
@ast
Robin R Ingalls
@en
Robin R Ingalls
@nl
prefLabel
Robin R Ingalls
@ast
Robin R Ingalls
@en
Robin R Ingalls
@nl
P106
P31
P496
0000-0003-2197-7198