about
Apc mutation enhances PyMT-induced mammary tumorigenesisThe Bloom's syndrome protein (BLM) interacts with MLH1 but is not required for DNA mismatch repairThe APC tumor suppressor is required for epithelial cell polarization and three-dimensional morphogenesisWnt/beta-catenin pathway activation is enriched in basal-like breast cancers and predicts poor outcome.APC/β-catenin-rich complexes at membrane protrusions regulate mammary tumor cell migration and mesenchymal morphologyβ-Catenin is required for the tumorigenic behavior of triple-negative breast cancer cells.β-catenin regulates c-Myc and CDKN1A expression in breast cancer cells.A Wnt-ow of opportunity: targeting the Wnt/beta-catenin pathway in breast cancer.The Ron receptor tyrosine kinase is not required for adenoma formation in Apc(Min/+) miceStrain-specific of alachlor on murine olfactory mucosal responses.The APC tumor suppressor is required for epithelial integrity in the mouse mammary gland.The APC tumor suppressor controls entry into S-phase through its ability to regulate the cyclin D/RB pathway.Enhanced tumor formation in mice heterozygous for Blm mutationDiffering effects of endogenous and synthetic inhibitors of metalloproteinases on intestinal tumorigenesis
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-7379-5639
@en
name
Kathleen H Goss
@ast
Kathleen H Goss
@en
Kathleen H Goss
@es
Kathleen H Goss
@nl
type
label
Kathleen H Goss
@ast
Kathleen H Goss
@en
Kathleen H Goss
@es
Kathleen H Goss
@nl
prefLabel
Kathleen H Goss
@ast
Kathleen H Goss
@en
Kathleen H Goss
@es
Kathleen H Goss
@nl
P214
P106
P1153
7006677432
P214
P31
P496
0000-0002-7379-5639
P735
P7859
lccn-no98130832