about
Kinase-mediated quasi-dimers of EGFRMultiplexed tracking of combinatorial genomic mutations in engineered cell populations.EGR1 and the ERK-ERF axis drive mammary cell migration in response to EGF.Deubiquitination of EGFR by Cezanne-1 contributes to cancer progression.Genome-wide mapping of mutations at single-nucleotide resolution for protein, metabolic and genome engineering.Quantitative Tracking of Combinatorially Engineered Populations with Multiplexed Binary Assemblies.EGFRvIV: a previously uncharacterized oncogenic mutant reveals a kinase autoinhibitory mechanism.Bacterial Recombineering: Genome Engineering via Phage-Based Homologous Recombination.Interfering with the Dimerization of the ErbB Receptors by Transmembrane Domain-Derived Peptides Inhibits Tumorigenic Growth in Vitro and in Vivo.The Resistome: A Comprehensive Database of Escherichia coli Resistance Phenotypes.Defective ubiquitinylation of EGFR mutants of lung cancer confers prolonged signaling.Evolutionary Genomics.SILAC identifies LAD1 as a filamin-binding regulator of actin dynamics in response to EGF and a marker of aggressive breast tumors.The ERBB network: at last, cancer therapy meets systems biology.Genomic Deoxyxylulose Phosphate Reductoisomerase (DXR) Mutations Conferring Resistance to the Antimalarial Drug Fosmidomycin in E. coliDynamic Management of Codon Compression for Saturation MutagenesisPredicting Drug Resistance Using Deep Mutational Scanning
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-1757-6722
@en
name
Gur Pines
@ast
Gur Pines
@en
Gur Pines
@es
Gur Pines
@nl
type
label
Gur Pines
@ast
Gur Pines
@en
Gur Pines
@es
Gur Pines
@nl
prefLabel
Gur Pines
@ast
Gur Pines
@en
Gur Pines
@es
Gur Pines
@nl
P106
P1153
22942241800
P31
P496
0000-0002-1757-6722