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The regulation of inflammation by interferons and their STATsRoute of Infection Determines the Impact of Type I Interferons on Innate Immunity to Listeria monocytogenesTracking heavy water (D2O) incorporation for identifying and sorting active microbial cells.Alarin is a vasoactive peptide.Intestinal Epithelial Cell Tyrosine Kinase 2 Transduces IL-22 Signals To Protect from Acute Colitis.Galanin message-associated peptide suppresses growth and the budded-to-hyphal-form transition of Candida albicansPhylotype-level 16S rRNA analysis reveals new bacterial indicators of health state in acute murine colitis.Intestinal Microbiota Signatures Associated with Inflammation History in Mice Experiencing Recurring Colitis.Host-compound foraging by intestinal microbiota revealed by single-cell stable isotope probing.NAIP proteins are required for cytosolic detection of specific bacterial ligands in vivo.Longitudinal study of murine microbiota activity and interactions with the host during acute inflammation and recovery.NAIP-NLRC4 Inflammasomes Coordinate Intestinal Epithelial Cell Expulsion with Eicosanoid and IL-18 Release via Activation of Caspase-1 and -8.Regulation of NO synthesis, local inflammation, and innate immunity to pathogens by BET family proteins.Noncanonical Effects of IRF9 in Intestinal Inflammation: More than Type I and Type III Interferons.Type I interferons have opposing effects during the emergence and recovery phases of colitis.Galanin is a modulator of eccrine sweat gland secretion.Evidence that the modulatory effect of galanin on inflammatory edema formation is mediated by the galanin receptor 3 in the murine microvasculature.Effects of galanin message-associated peptide and neuropeptide Y against various non-albicans Candida strainsThe Galanin System in CancerAnti-Candida activity of α-melanocyte-stimulating hormone (α-MSH) peptidesMMP-9 haplotypes and carotid artery atherosclerosis: an association study introducing a novel multicolour multiplex RealTime PCR protocolIntestinal restriction of Salmonella Typhimurium requires caspase-1 and caspase-11 epithelial intrinsic inflammasomesTranscriptional profiling identifies caspase-1 as a T cell-intrinsic regulator of Th17 differentiationGasdermin-D and Caspase-7 are the key Caspase-1/8 substrates downstream of the NAIP5/NLRC4 inflammasome required for restriction of Legionella pneumophilaNLRC4 inflammasome activation is NLRP3- and phosphorylation-independent during infection and does not protect from melanoma
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description
researcher ORCID ID = 0000-0003-1897-5693
@en
wetenschapper
@nl
name
Isabella Rauch
@ast
Isabella Rauch
@en
Isabella Rauch
@es
Isabella Rauch
@nl
type
label
Isabella Rauch
@ast
Isabella Rauch
@en
Isabella Rauch
@es
Isabella Rauch
@nl
prefLabel
Isabella Rauch
@ast
Isabella Rauch
@en
Isabella Rauch
@es
Isabella Rauch
@nl
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17346923000
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P31
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0000-0003-1897-5693