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Edaravone protects against methylglyoxal-induced barrier damage in human brain endothelial cellsTesting the neurovascular hypothesis of Alzheimer's disease: LRP-1 antisense reduces blood-brain barrier clearance, increases brain levels of amyloid-beta protein, and impairs cognitionHuman immunodeficiency virus-1 uses the mannose-6-phosphate receptor to cross the blood-brain barrierIn vitro blood-brain barrier models using brain capillary endothelial cells isolated from neonatal and adult rats retain age-related barrier properties.Lipopolysaccharide-enhanced transcellular transport of HIV-1 across the blood-brain barrier is mediated by luminal microvessel IL-6 and GM-CSF.Alpha Adrenergic Induction of Transport of Lysosomal Enzyme across the Blood-Brain Barrier.Contribution of thrombin-reactive brain pericytes to blood-brain barrier dysfunction in an in vivo mouse model of obesity-associated diabetes and an in vitro rat modelNitric oxide isoenzymes regulate lipopolysaccharide-enhanced insulin transport across the blood-brain barrier.Lipopolysaccharide-enhanced transcellular transport of HIV-1 across the blood-brain barrier is mediated by the p38 mitogen-activated protein kinase pathwayBrain pericytes increase the lipopolysaccharide-enhanced transcytosis of HIV-1 free virus across the in vitro blood-brain barrier: evidence for cytokine-mediated pericyte-endothelial cell crosstalkLipopolysaccharide alters the blood-brain barrier transport of amyloid beta protein: a mechanism for inflammation in the progression of Alzheimer's diseaseBrain pericytes among cells constituting the blood-brain barrier are highly sensitive to tumor necrosis factor-α, releasing matrix metalloproteinase-9 and migrating in vitroBrain pericyte-derived soluble factors enhance insulin sensitivity in GT1-7 hypothalamic neurons.Compounds blocking methylglyoxal-induced protein modification and brain endothelial injury.Paracellular barrier and tight junction protein expression in the immortalized brain endothelial cell lines bEND.3, bEND.5 and mouse brain endothelial cell 4.Partial hepatectomy aggravates cyclosporin A-induced neurotoxicity by lowering the function of the blood-brain barrier in mice.Adverse effect of cyclosporin A on barrier functions of cerebral microvascular endothelial cells after hypoxia-reoxygenation damage in vitro.Brain pericytes contribute to the induction and up-regulation of blood-brain barrier functions through transforming growth factor-beta production.Nitric oxide mediates cyclosporine-induced impairment of the blood-brain barrier in cocultures of mouse brain endothelial cells and rat astrocytes.Transforming growth factor-beta1 upregulates the tight junction and P-glycoprotein of brain microvascular endothelial cells.Uptake and efflux of quinacrine, a candidate for the treatment of prion diseases, at the blood-brain barrier.Cyclophilin A secreted from fibroblast-like synoviocytes is involved in the induction of CD147 expression in macrophages of mice with collagen-induced arthritis.Detachment of brain pericytes from the basal lamina is involved in disruption of the blood-brain barrier caused by lipopolysaccharide-induced sepsis in mice.Cyclosporin A induces hyperpermeability of the blood-brain barrier by inhibiting autocrine adrenomedullin-mediated up-regulation of endothelial barrier function.Tumor necrosis factor-alpha mediates the blood-brain barrier dysfunction induced by activated microglia in mouse brain microvascular endothelial cells.Lipopolysaccharide-activated microglia induce dysfunction of the blood-brain barrier in rat microvascular endothelial cells co-cultured with microglia.Contrast media increase vascular endothelial permeability by inhibiting nitric-oxide production.Subchronic treatment with cyclosporin A decreases the binding properties of the GABAA receptor in ovariectomized rats.Cyclosporine A-increased nitric oxide production in the rat dorsal hippocampus mediates convulsions.Adrenomedullin-induced relaxation of rat brain pericytes is related to the reduced phosphorylation of myosin light chain through the cAMP/PKA signaling pathway.Cyclosporin A aggravates electroshock-induced convulsions in mice with a transient middle cerebral artery occlusion.Peroxisome proliferator-activated receptor-gamma-mediated positive energy balance in the rat is associated with reduced sympathetic drive to adipose tissues and thyroid status.A role for hypothalamic AMP-activated protein kinase in the mediation of hyperphagia and weight gain induced by chronic treatment with olanzapine in female rats.Metformin induces up-regulation of blood-brain barrier functions by activating AMP-activated protein kinase in rat brain microvascular endothelial cells.Disruption of the blood-brain barrier in collagen-induced arthritic mice.Copper complexing decreases the ability of amyloid beta peptide to cross the BBB and enter brain parenchyma.Brain pericytes are the most thrombin-sensitive matrix metalloproteinase-9-releasing cell type constituting the blood-brain barrier in vitro.Role of thrombin-PAR1-PKCθ/δ axis in brain pericytes in thrombin-induced MMP-9 production and blood-brain barrier dysfunction in vitro.An inhibitory role of nitric oxide in the dynamic regulation of the blood-brain barrier function.Inhibition of transforming growth factor-beta production in brain pericytes contributes to cyclosporin A-induced dysfunction of the blood-brain barrier.
P50
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P50
description
researcher ORCID ID = 0000-0002-8036-0510
@en
name
Shinya Dohgu
@ast
Shinya Dohgu
@en
Shinya Dohgu
@es
Shinya Dohgu
@nl
type
label
Shinya Dohgu
@ast
Shinya Dohgu
@en
Shinya Dohgu
@es
Shinya Dohgu
@nl
prefLabel
Shinya Dohgu
@ast
Shinya Dohgu
@en
Shinya Dohgu
@es
Shinya Dohgu
@nl
P31
P496
0000-0002-8036-0510