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Modulation of T-cell activation by the glucocorticoid-induced leucine zipper factor via inhibition of nuclear factor kappaBA new dexamethasone-induced gene of the leucine zipper family protects T lymphocytes from TCR/CD3-activated cell deathSynthesis of glucocorticoid-induced leucine zipper (GILZ) by macrophages: an anti-inflammatory and immunosuppressive mechanism shared by glucocorticoids and IL-10.GITR, a member of the TNF receptor superfamily, is costimulatory to mouse T lymphocyte subpopulations.Molecular mechanisms of immunomodulatory activity of glucocorticoids.Glucocorticoid-induced leucine zipper (GILZ) over-expression in T lymphocytes inhibits inflammation and tissue damage in spinal cord injury.Silymarin suppress CD4+ T cell activation and proliferation: effects on NF-kappaB activity and IL-2 production.Glucocorticoid-induced leucine zipper (GILZ)/NF-kappaB interaction: role of GILZ homo-dimerization and C-terminal domain.Synthesis and evaluation of anti-apoptotic activity of L-carnitine cyclic analogues and amino acid derivatives.Lack of glucocorticoid-induced leucine zipper (GILZ) deregulates B-cell survival and results in B-cell lymphocytosis in mice.GITR modulates innate and adaptive mucosal immunity during the development of experimental colitis in mice.Induction of apoptosis by 1,4-benzothiazine analogs in mouse thymocytes.Apoptosis of human primary B lymphocytes is inhibited by N-acetyl-L-cysteine.Role of glucocorticoid-induced TNF receptor family gene (GITR) in collagen-induced arthritis.Cloning, chromosomal assignment and tissue distribution of human GILZ, a glucocorticoid hormone-induced gene.Endothelial dysfunction in vivo is related to monocyte resistin mRNA expression.Glucocorticoid-Induced Leucine Zipper (GILZ) Controls Inflammation and Tissue Damage after Spinal Cord InjuryDifferentiation of Ly49s-positive or -negative natural killer cells is inhibited by anti-H-2b monoclonal antibodies acting at the level of bone marrow progenitors from B6 miceEffect of interleukin-2 on generation of natural killer cells: role of major histocompatibility complex class I in B6 and TAP-1-/- miceIL-2-driven natural killer cell generation: role of anti-H-2b monoclonal antibodies and stromal cells in controlling quantitative and repertoire changesGILZ, a glucocorticoid hormone induced gene, modulates T lymphocytes activation and death through interaction with NF-kBGenomic and non-genomic effects of different glucocorticoids on mouse thymocyte apoptosisGITR gene deletion and GITR-FC soluble protein administration inhibit multiple organ failure induced by zymosan
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description
researcher ORCID ID = 0000-0003-0313-1615
@en
name
Stefano Bruscoli
@ast
Stefano Bruscoli
@en
Stefano Bruscoli
@es
Stefano Bruscoli
@nl
type
label
Stefano Bruscoli
@ast
Stefano Bruscoli
@en
Stefano Bruscoli
@es
Stefano Bruscoli
@nl
prefLabel
Stefano Bruscoli
@ast
Stefano Bruscoli
@en
Stefano Bruscoli
@es
Stefano Bruscoli
@nl
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P31
P496
0000-0003-0313-1615