about
Fetal cells traffic to injured maternal myocardium and undergo cardiac differentiation.JAK2 inhibitors do not affect stem cells present in the spleens of patients with myelofibrosis.Sequential treatment of CD34+ cells from patients with primary myelofibrosis with chromatin-modifying agents eliminate JAK2V617F-positive NOD/SCID marrow repopulating cells.Combination treatment in vitro with Nutlin, a small-molecule antagonist of MDM2, and pegylated interferon-α 2a specifically targets JAK2V617F-positive polycythemia vera cellsReappraising hyalinizing clear cell carcinoma: A population-based study with molecular confirmation.Analysis of ALK gene in 133 patients with breast cancer revealed polysomy of chromosome 2 and no ALK amplification.Double minute amplification of mutant PDGF receptor α in a mouse glioma model.BCR-ABL1 is a secondary event after JAK2V617F in patients with polycythemia vera who develop chronic myeloid leukemia.Genomic characterization of spleens in patients with myelofibrosisAdvanced forms of MPNs are accompanied by chromosomal abnormalities that lead to dysregulation of TP53
P50
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P50
description
researcher ORCID ID = 0000-0003-3033-1081
@en
name
Joseph Tripodi
@ast
Joseph Tripodi
@en
Joseph Tripodi
@es
Joseph Tripodi
@nl
type
label
Joseph Tripodi
@ast
Joseph Tripodi
@en
Joseph Tripodi
@es
Joseph Tripodi
@nl
prefLabel
Joseph Tripodi
@ast
Joseph Tripodi
@en
Joseph Tripodi
@es
Joseph Tripodi
@nl
P31
P496
0000-0003-3033-1081