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Novel Findings from CNVs Implicate Inhibitory and Excitatory Signaling Complexes in SchizophreniaClustered coding variants in the glutamate receptor complexes of individuals with schizophrenia and bipolar disorderGenetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's diseaseDe novo CNV analysis implicates specific abnormalities of postsynaptic signalling complexes in the pathogenesis of schizophrenia.Biological overlap of attention-deficit/hyperactivity disorder and autism spectrum disorder: evidence from copy number variantsDe novo mutations in schizophrenia implicate synaptic networksExome arrays capture polygenic rare variant contributions to schizophrenia.Gender differences in CNV burden do not confound schizophrenia CNV associations.Analysis of exome sequence in 604 trios for recessive genotypes in schizophrenia.The synapse in schizophrenia.Contribution of copy number variants to schizophrenia from a genome-wide study of 41,321 subjects.Analysis of Intellectual Disability Copy Number Variants for Association With Schizophrenia.Evolutionary expansion and anatomical specialization of synapse proteome complexity.Mutation intolerant genes and targets of FMRP are enriched for nonsynonymous alleles in schizophrenia.Neurotransmitters drive combinatorial multistate postsynaptic density networks.Treating the placenta to prevent adverse effects of gestational hypoxia on fetal brain development.Schizophrenia copy number variants and associative learningReplication of brain function effects of a genome-wide supported psychiatric risk variant in the CACNA1C gene and new multi-locus effects.Arc Requires PSD95 for Assembly into Postsynaptic Complexes Involved with Neural Dysfunction and Intelligence.Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection.Structural and Functional Neuroimaging of Polygenic Risk for Schizophrenia: A Recall-by-Genotype-Based Approach.Correction: Genetic Evidence Implicates the Immune System and Cholesterol Metabolism in the Aetiology of Alzheimer's Disease.Common schizophrenia alleles are enriched in mutation-intolerant genes and maintained by background selectionA contribution of novel CNVs to schizophrenia from a genome-wide study of 41,321 subjectsDynamic expression of genes associated with schizophrenia and bipolar disorder across developmentCortical patterning of abnormal morphometric similarity in psychosis is associated with brain expression of schizophrenia-related genesCortical patterning of abnormal morphometric similarity in psychosis is associated with brain expression of schizophrenia related genes: Supplementary InformationNovel Insight into the Aetiology of Autism Spectrum Disorder Gained by Integrating Expression Data with Genome-wide Association StatisticsTargeted Sequencing of 10,198 Samples Confirms Abnormalities in Neuronal Activity and Implicates Voltage-Gated Sodium Channels in Schizophrenia PathogenesisPredictive modeling of schizophrenia from genomic data: Comparison of polygenic risk score with kernel support vector machines approachDe novo mutations identified by exome sequencing implicate rare missense variants in SLC6A1 in schizophrenia
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researcher ORCID ID = 0000-0002-2137-0452
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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Andrew Pocklington
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0000-0002-2137-0452