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Over the counter (OTC) artificial tear drops for dry eye syndromeThe ability of inner-cell-mass cells to self-renew as embryonic stem cells is acquired following epiblast specificationRegulated fluctuations in nanog expression mediate cell fate decisions in embryonic stem cellsPromotion of reprogramming to ground state pluripotency by signal inhibitionThe Stat6-regulated KRAB domain zinc finger protein Zfp157 regulates the balance of lineages in mammary glands and compensates for loss of Gata-3Alpha-fetoprotein, the major fetal serum protein, is not essential for embryonic development but is required for female fertility.Loss of Wnt8b has no overt effect on hippocampus development but leads to altered Wnt gene expression levels in dorsomedial telencephalonThe NuRD component Mbd3 is required for pluripotency of embryonic stem cellsMbd3, a component of the NuRD co-repressor complex, is required for development of pluripotent cellsNanog safeguards pluripotency and mediates germline developmentEmbryonic germ cells from mice and rats exhibit properties consistent with a generic pluripotent ground state.Tracking the embryonic stem cell transition from ground state pluripotency.Primate embryogenesis predicts the hallmarks of human naïve pluripotency.Isolation and propagation of enteric neural crest progenitor cells from mouse embryonic stem cells and embryosThe many faces of Pluripotency: in vitro adaptations of a continuum of in vivo states.Impact of a rinse step on protein removal from silicone hydrogel contact lenses.Resetting transcription factor control circuitry toward ground-state pluripotency in human.A practical and efficient cellular substrate for the generation of induced pluripotent stem cells from adults: blood-derived endothelial progenitor cellsTotipotent embryonic stem cells arise in ground-state culture conditions.NANOG amplifies STAT3 activation and they synergistically induce the naive pluripotent program.LIF-independent JAK signalling to chromatin in embryonic stem cells uncovered from an adult stem cell disease.The birth of embryonic pluripotency.A conserved Oct4/POUV-dependent network links adhesion and migration to progenitor maintenanceThe nonconventional MHC class II molecule DM governs diabetes susceptibility in NOD miceKlf4 reverts developmentally programmed restriction of ground state pluripotencyThe opposing transcriptional functions of Sin3a and c-Myc are required to maintain tissue homeostasis.Human hypoblast formation is not dependent on FGF signalling.The germ cell determinant Blimp1 is not required for derivation of pluripotent stem cells.Acute loss of Cited2 impairs Nanog expression and decreases self-renewal of mouse embryonic stem cellsThe transcriptional and epigenomic foundations of ground state pluripotency.Pluripotency in the embryo and in cultureGermline potential of parthenogenetic haploid mouse embryonic stem cells.Nanog is the gateway to the pluripotent ground state.Lineage-Specific Profiling Delineates the Emergence and Progression of Naive Pluripotency in Mammalian EmbryogenesisA SNP in the Immunoregulatory Molecule CTLA-4 Controls mRNA Splicing In Vivo but Does Not Alter Diabetes Susceptibility in the NOD MouseNaive Pluripotent Stem Cells Derived Directly from Isolated Cells of the Human Inner Cell MassRebuilding pluripotency from primordial germ cells.Suppression of Erk signalling promotes ground state pluripotency in the mouse embryo.Oct4 is required for lineage priming in the developing inner cell mass of the mouse blastocyst.Overcoming self-destruction in the pancreas.
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description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-8650-1388
@en
name
Jennifer Nichols
@ast
Jennifer Nichols
@en
Jennifer Nichols
@es
Jennifer Nichols
@nl
type
label
Jennifer Nichols
@ast
Jennifer Nichols
@en
Jennifer Nichols
@es
Jennifer Nichols
@nl
prefLabel
Jennifer Nichols
@ast
Jennifer Nichols
@en
Jennifer Nichols
@es
Jennifer Nichols
@nl
P108
P106
P21
P31
P496
0000-0002-8650-1388