about
Complex transcriptional effects of p63 isoforms: identification of novel activation and repression domainsHeterozygous germline mutations in the p53 homolog p63 are the cause of EEC syndromeMad2 is a critical mediator of the chromosome instability observed upon Rb and p53 pathway inhibition.In epithelial cancers, aberrant COL17A1 promoter methylation predicts its misexpression and increased invasion.The cancer biology of whole-chromosome instability.A novel translation re-initiation mechanism for the p63 gene revealed by amino-terminal truncating mutations in Rapp-Hodgkin/Hay-Wells-like syndromes.p63 Gene mutations in eec syndrome, limb-mammary syndrome, and isolated split hand-split foot malformation suggest a genotype-phenotype correlation.Silencing of BACH1 inhibits invasion and migration of prostate cancer cells by altering metastasis-related gene expression.Translocation Breakpoints Preferentially Occur in Euchromatin and Acrocentric Chromosomes.Overexpression of Ran GTPase Components Regulating Nuclear Export, but not Mitotic Spindle Assembly, Marks Chromosome Instability and Poor Prognosis in Breast Cancer.In vivo overexpression of Emi1 promotes chromosome instability and tumorigenesis.Delineation of the ADULT syndrome phenotype due to arginine 298 mutations of the p63 geneContradictory mRNA and protein misexpression of EEF1A1 in ductal breast carcinoma due to cell cycle regulation and cellular stressCEP55 is a determinant of cell fate during perturbed mitosis in breast cancerTreating cancer with microRNA replacement therapy: A literature reviewMicroRNAs in cancer drug resistance: Basic evidence and clinical applicationsMitotic slippage: an old tale with a new twist.The role of miR-34 in cancer drug resistancemicroRNAs in cancer stem cells: Biology, pathways, and therapeutic opportunitiesDownregulation of miR-146a promotes cell migration in Helicobacter pylori-negative gastric cancerA functional genetic screen identifies the Mediator complex as essential for SSX2-induced senescencePatterns of Genomic Instability in Breast CancermiR-142-3p is a tumor suppressor that inhibits estrogen receptor expression in ER-positive breast cancerCommonly integrated epigenetic modifications of differentially expressed genes lead to adaptive resistance in cancerMechanisms of Genomic Instability in Breast CancerHMGA2 and Bach-1 cooperate to promote breast cancer cell malignancyMicroRNAs in cancer cell death pathways: Apoptosis and necroptosisInteractions between cancer stem cells, immune system and some environmental components: Friends or foes?Multi-Omics Characterization of the Spontaneous Mesenchymal-Epithelial Transition in the PMC42 Breast Cancer Cell LinesChromosome arm aneuploidies shape tumour evolution and drug responseCorrigendum to "MicroRNAs in cancer cell death pathways: Apoptosis and necroptosis" [Free Radic. Biol. Med. 139 (2019) 1-15]miR-330 suppresses EMT and induces apoptosis by downregulating HMGA2 in human colorectal cancermiR-142-3p as tumor suppressor miRNA in the regulation of tumorigenicity, invasion and migration of human breast cancer by targeting Bach-1 expressionComplexities of pharmacogenomic interactions in cancerBaseline pulmonary levels of CD8+ T cells and NK cells inversely correlate with expression of the SARS-CoV-2 entry receptor ACE2
P50
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P50
description
researcher ORCID ID = 0000-0001-8646-9843
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name
Pascal Hg Duijf
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Pascal Hg Duijf
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Pascal Hg Duijf
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Pascal Hg Duijf
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type
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Pascal Hg Duijf
@ast
Pascal Hg Duijf
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Pascal Hg Duijf
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Pascal Hg Duijf
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Pascal Hg Duijf
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Pascal Hg Duijf
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Pascal Hg Duijf
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Pascal Hg Duijf
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P108
P108
P1153
6506948308
P31
P496
0000-0001-8646-9843