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Modulation of Alloimmunity by Heat Shock ProteinsThe Scavenger Receptor SREC-I Cooperates with Toll-Like Receptors to Trigger Inflammatory Innate Immune ResponsesMolecular stress-inducing compounds increase osteoclast formation in a heat shock factor 1 protein-dependent mannerHeat-shock factor 1 both positively and negatively affects cellular clonogenic growth depending on p53 status.Histone deacetylase activity mediates acquired resistance towards structurally diverse HSP90 inhibitors.A Workflow Guide to RNA-seq Analysis of Chaperone Function and Beyond.HSP90 inhibitors enhance differentiation and MITF (microphthalmia transcription factor) activity in osteoclast progenitors.March1-dependent modulation of donor MHC II on CD103 dendritic cells mitigates alloimmunityGenotoxic stress induces Sca-1-expressing metastatic mammary cancer cells.Heat Shock Proteins Are Essential Components in Transformation and Tumor Progression: Cancer Cell Intrinsic Pathways and BeyondCell Stress Induced Stressome Release Including Damaged Membrane Vesicles and Extracellular HSP90 by Prostate Cancer CellsMZF1 and SCAND1 Reciprocally Regulate CDC37 Gene Expression in Prostate CancerImmunological Outcomes Mediated Upon Binding of Heat Shock Proteins to Scavenger Receptors SCARF1 and LOX-1, and Endocytosis by Mononuclear PhagocytesHSF1: Primary Factor in Molecular Chaperone Expression and a Major Contributor to Cancer Morbidity
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P50
description
researcher ORCID ID = 0000-0002-2205-8591
@en
name
Benjamin J Lang
@ast
Benjamin J Lang
@en
Benjamin J Lang
@es
Benjamin J Lang
@nl
type
label
Benjamin J Lang
@ast
Benjamin J Lang
@en
Benjamin J Lang
@es
Benjamin J Lang
@nl
prefLabel
Benjamin J Lang
@ast
Benjamin J Lang
@en
Benjamin J Lang
@es
Benjamin J Lang
@nl
P1153
55308999200
P31
P496
0000-0002-2205-8591