about
Aberrant excitatory neuronal activity and compensatory remodeling of inhibitory hippocampal circuits in mouse models of Alzheimer's diseaseLong-lasting adaptations of the NR2B-containing NMDA receptors in the dorsomedial striatum play a crucial role in alcohol consumption and relapse.Mind bomb-2 is an E3 ligase that ubiquitinates the N-methyl-D-aspartate receptor NR2B subunit in a phosphorylation-dependent manner.Striatal involvement in human alcoholism and alcohol consumption, and withdrawal in animal modelsAlcohol Elicits Functional and Structural Plasticity Selectively in Dopamine D1 Receptor-Expressing Neurons of the Dorsomedial Striatum.Ethanol-mediated facilitation of AMPA receptor function in the dorsomedial striatum: implications for alcohol drinking behavior.Protein tyrosine phosphatase α in the dorsomedial striatum promotes excessive ethanol-drinking behaviors.Dopamine D1 or D2 receptor-expressing neurons in the central nervous system.Glial cell line-derived neurotrophic factor (GDNF) is an endogenous protector in the mesolimbic system against excessive alcohol consumption and relapse.Ethanol alters trafficking and functional N-methyl-D-aspartate receptor NR2 subunit ratio via H-Ras.GDNF acutely potentiates Ca2+ channels and excitatory synaptic transmission in midbrain dopaminergic neurons.Splice variants of the NR1 subunit differentially induce NMDA receptor-dependent gene expression.Ethanol induces long-term facilitation of NR2B-NMDA receptor activity in the dorsal striatum: implications for alcohol drinking behavior.Bidirectional and long-lasting control of alcohol-seeking behavior by corticostriatal LTP and LTD.Prenatal Exposure to Alcohol Induces Functional and Structural Plasticity in Dopamine D1 Receptor-Expressing Neurons of the Dorsomedial StriatumAlcohol intake enhances glutamatergic transmission from D2 receptor-expressing afferents onto D1 receptor-expressing medium spiny neurons in the dorsomedial striatumOptogenetic control of alcohol-seeking behavior via the dorsomedial striatal circuitThe use of chemogenetic approaches in alcohol use disorder research and treatment
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description
researcher ORCID ID = 0000-0002-0085-4722
@en
name
Jun Wang
@ast
Jun Wang
@en
Jun Wang
@es
Jun Wang
@nl
type
label
Jun Wang
@ast
Jun Wang
@en
Jun Wang
@es
Jun Wang
@nl
prefLabel
Jun Wang
@ast
Jun Wang
@en
Jun Wang
@es
Jun Wang
@nl
P31
P496
0000-0002-0085-4722