about
Bayesian and maximum likelihood phylogenetic analyses of protein sequence data under relative branch-length differences and model violationSingle-cell transcriptogenomics reveals transcriptional exclusion of ENU-mutated allelesThe meta-epigenomic structure of purified human stem cell populations is defined at cis-regulatory sequencesTherapeutic implications of GIPC1 silencing in cancerData-driven normalization strategies for high-throughput quantitative RT-PCR.Decomposition of gene expression state space trajectoriesAllele-specific genome-wide profiling in human primary erythroblasts reveal replication program organizationTemporal dynamics and transcriptional control using single-cell gene expression analysisDefining an informativeness metric for clustering gene expression dataVariance of gene expression identifies altered network constraints in neurological disease.Divergent reprogramming routes lead to alternative stem-cell states.attract: A method for identifying core pathways that define cellular phenotypes.nEASE: a method for gene ontology subclassification of high-throughput gene expression data.Gene expression variability as a unifying element of the pluripotency network.Viral perturbations of host networks reflect disease etiology.A promoter-level mammalian expression atlasEpstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines.Inferring steady state single-cell gene expression distributions from analysis of mesoscopic samplesThe transcriptional network that controls growth arrest and differentiation in a human myeloid leukemia cell line.Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth.Variability of Gene Expression Identifies Transcriptional Regulators of Early Human Embryonic Development.Genome-wide siRNA screen for mediators of NF-κB activationApplication of Gene Expression Trajectories Initiated from ErbB Receptor Activation Highlights the Dynamics of Divergent Promoter Usage.Chromosome instability in diffuse large B cell lymphomas is suppressed by activation of the noncanonical NF-κB pathway.Arteriolar niches maintain haematopoietic stem cell quiescence.Self-renewal of a purified Tie2+ hematopoietic stem cell population relies on mitochondrial clearance.Not to be suppressed? Rethinking the host response at a root-parasite interface.Changes in gene expression variability reveal a stable synthetic lethal interaction network in BRCA2- ovarian cancers.Not just a colourful metaphor: modelling the landscape of cellular development using Hopfield networks.pathVar: a new method for pathway-based interpretation of gene expression variability.Single-cell gene expression profiles define self-renewing, pluripotent, and lineage primed states of human pluripotent stem cells.HPV16 E7 oncogene expression in normal human epithelial cells causes molecular changes indicative of an epithelial to mesenchymal transition.Genome-wide characterization of the routes to pluripotency.Canonical NF-kappaB activation is essential for Epstein-Barr virus latent membrane protein 1 TES2/CTAR2 gene regulation.Metformin regulates metabolic and nonmetabolic pathways in skeletal muscle and subcutaneous adipose tissues of older adults.Further evidence for BRCA1 communication with the inactive X chromosome.Stem cell factor is selectively secreted by arterial endothelial cells in bone marrow.Evaluating methods of inferring gene regulatory networks highlights their lack of performance for single cell gene expression data.Erratum: Corrigendum: Divergent reprogramming routes lead to alternative stem-cell statesErratum: Corrigendum: Genome-wide characterization of the routes to pluripotency
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description
researcher ORCID ID = 0000-0002-5147-9299
@en
wetenschapper
@nl
name
Jessica C Mar
@ast
Jessica C Mar
@en
Jessica C Mar
@es
Jessica C Mar
@nl
type
label
Jessica C Mar
@ast
Jessica C Mar
@en
Jessica C Mar
@es
Jessica C Mar
@nl
altLabel
Jessica Mar
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prefLabel
Jessica C Mar
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Jessica C Mar
@en
Jessica C Mar
@es
Jessica C Mar
@nl
P106
P1153
8300771200
P21
P31
P496
0000-0002-5147-9299