about
The p38 MAPK pathway inhibits tristetraprolin-directed decay of interleukin-10 and pro-inflammatory mediator mRNAs in murine macrophagesGraded phenotypic response to partial and complete deficiency of a brain-specific transcript variant of the winged helix transcription factor RFX4MARCKS regulates membrane targeting of Rab10 vesicles to promote axon development.Hippocampal infusions of MARCKS peptides impair memory of rats on the radial-arm mazeThe RNA-binding protein, ZFP36L2, influences ovulation and oocyte maturation.Mitogen-activated protein kinase-activated protein kinase 2 regulates tumor necrosis factor mRNA stability and translation mainly by altering tristetraprolin expression, stability, and binding to adenine/uridine-rich elementDifferential post-transcriptional regulation of IL-10 by TLR2 and TLR4-activated macrophagesLPS-induced production of TNF-α and IL-6 in mast cells is dependent on p38 but independent of TTPTristetraprolin mediates interferon-gamma mRNA decay.The RNA-binding protein TTP is a global post-transcriptional regulator of feedback control in inflammationTristetraprolin is required for full anti-inflammatory response of murine macrophages to IL-10.Third Report on Chicken Genes and Chromosomes 2015.Interferons limit inflammatory responses by induction of tristetraprolin.Implantable drug-delivery systems / Perry J. Blackshear. - (1979)Transmembrane TNF protects mutant mice against intracellular bacterial infections, chronic inflammation and autoimmunity.
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description
Amerikaans arts
@nl
Arzt und Forscher
@de
researcher ORCID ID = 0000-0002-9561-8529
@en
name
Perry J. Blackshear
@ast
Perry J. Blackshear
@de
Perry J. Blackshear
@en
Perry J. Blackshear
@nl
type
label
Perry J. Blackshear
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Perry J. Blackshear
@de
Perry J. Blackshear
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Perry J. Blackshear
@nl
altLabel
P. J. Blackshear
@de
PJ Blackshear
@de
Perry Blackshear
@de
prefLabel
Perry J. Blackshear
@ast
Perry J. Blackshear
@de
Perry J. Blackshear
@en
Perry J. Blackshear
@nl
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0000-0002-9561-8529