about
Pharmacological strategies to contend against myocardial reperfusion damage: diverse chemicals for multiple targets.Targeting mitochondria for cardiac protection.Redox activation of Nrf2 & NF-κB: a double end sword?Protective behavior of tamoxifen against Hg2+-induced toxicity on kidney mitochondria: in vitro and in vivo experiments.Mitochondrial inactivation by Anopheles albimanus cecropin 3: molecular mechanisms.Cellular stress associated with the differentiation of Plasmodium berghei ookinetes.Citicoline (CDP-choline) protects myocardium from ischemia/reperfusion injury via inhibiting mitochondrial permeability transition.Tamoxifen inhibits mitochondrial oxidative stress damage induced by copper orthophenanthroline.Role of sphingomyelinase in mitochondrial ceramide accumulation during reperfusion.Cardioprotection by curcumin post-treatment in rats with established chronic kidney disease.Antiarrhythmic effect of tamoxifen on the vulnerability induced by hyperthyroidism to heart ischemia/reperfusion damage.Cardioprotective kinase signaling to subsarcolemmal and interfibrillar mitochondria is mediated by caveolar structures.3-NP-induced Huntington's-like disease impairs Nrf2 activation without loss of cardiac function in aged rats.CDP-choline protects liver from ischemia/reperfusion injury preserving mitochondrial function and reducing oxidative stress.Cardioprotective properties of citicoline against hyperthyroidism-induced reperfusion damage in rat hearts.Inhibition of the nitric oxide/cyclic guanosine monophosphate pathway limited the cardioprotective effect of post-conditioning in hearts with apical myocardial infarction.Tamoxifen inhibits mitochondrial membrane damage caused by disulfiram.Nrf2-regulated antioxidant response is activated by protein kinase C in postconditioned rat hearts.On the Oxidative Damage by Cadmium on Kidney Mitochondrial FunctionsCurcumin maintains cardiac and mitochondrial function in chronic kidney diseaseAttenuation of oxidant damage in the postconditioned heart involves non-enzymatic response and partial catalytic protectionOn the properties of calcium-induced permeability transition in neonatal heart mitochondriaProtective effect of α-mangostin on cardiac reperfusion damage by attenuation of oxidative stress.Ebselen induces mitochondrial permeability transition because of its interaction with adenine nucleotide translocaseRedox signaling in ischemic postconditioning protection involves PKCε and Erk1/2 pathways and converges indirectly in Nrf2 activationCalcium Induces Mitochondrial Oxidative Stress Because of its Binding to Adenine Nucleotide TranslocaseAdipogenesis: A Necessary but Harmful Strategy
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Mabel Buelna-Chontal
@ast
Mabel Buelna-Chontal
@en
Mabel Buelna-Chontal
@es
Mabel Buelna-Chontal
@nl
type
label
Mabel Buelna-Chontal
@ast
Mabel Buelna-Chontal
@en
Mabel Buelna-Chontal
@es
Mabel Buelna-Chontal
@nl
prefLabel
Mabel Buelna-Chontal
@ast
Mabel Buelna-Chontal
@en
Mabel Buelna-Chontal
@es
Mabel Buelna-Chontal
@nl
P106
P31
P496
0000-0002-9554-9412