about
Caspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death.Genome-wide mutant fitness profiling identifies nutritional requirements for optimal growth of Yersinia pestis in deep tissue.Fibrin microthreads support mesenchymal stem cell growth while maintaining differentiation potentialThe Yersinia pestis Effector YopM Inhibits Pyrin Inflammasome Activation.Reversion From Methicillin Susceptibility to Methicillin Resistance in Staphylococcus aureus During Treatment of Bacteremia.Manipulation of Interleukin-1β and Interleukin-18 Production by Yersinia pestis Effectors YopJ and YopM and Redundant Impact on Virulence.Tuberculosis Susceptibility and Vaccine Protection Are Independently Controlled by Host GenotypeManipulation of interleukin-1β and interleukin-18 production by Yersinia pestis effectors YopJ and YopM and redundant impact on virulence.The NLRP12 inflammasome recognizes Yersinia pestis.Reply to Gelfand and Cleveland.Pathogen blockade of TAK1 triggers caspase-8-dependent cleavage of gasdermin D and cell deathThe NLRP12 Inflammasome Recognizes Yersinia pestisGain-of-Function Analysis Reveals Important Virulence Roles for the Yersinia pestis Type III Secretion System Effectors YopJ, YopT, and YpkA.Functionally Overlapping Variants Control Tuberculosis Susceptibility in Collaborative Cross MiceRedundant and Cooperative Roles for Yersinia pestis Yop Effectors in the Inhibition of Human Neutrophil Exocytic Responses Revealed by Gain-of-Function ApproachCommon Variants in the Glycerol Kinase Gene Reduce Tuberculosis Drug EfficacyLarge-scale chemical-genetics yields new M. tuberculosis inhibitor classes
P50
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P50
description
auteur
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hulumtuese
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հետազոտող
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Megan K Proulx
@nl
Megan K. Proulx
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Megan K. Proulx
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type
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Megan K Proulx
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Megan K. Proulx
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Megan K. Proulx
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M K Proulx
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M. K. Proulx
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Megan K Proulx
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Megan Proulx
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Megan K Proulx
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Megan K. Proulx
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Megan K. Proulx
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